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	<title>DEFINITIVE MIND &#187; Psychiatry</title>
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	<link>http://www.definitivemind.com</link>
	<description>Optimizing Mental and Physical Health through the Fusion of Knowledge and Skills from Neuroscience, Psychology, Psychiatry, Neuroendocrinology, Psychoneuroimmunology, Biochemistry, Nutritional Science and Other Fields in Art, Medicine, Philosophy, Science and Technology.  By Romeo Mariano MD</description>
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		<title>Astrocytes Use ATP as a Signal to Regulate Synaptic Plasticity Of Neural Circuits</title>
		<link>http://www.definitivemind.com/2012/10/14/astrocytes-use-atp-as-a-signal-to-regulate-synaptic-plasticity-of-neural-circuits/</link>
		<comments>http://www.definitivemind.com/2012/10/14/astrocytes-use-atp-as-a-signal-to-regulate-synaptic-plasticity-of-neural-circuits/#comments</comments>
		<pubDate>Sun, 14 Oct 2012 20:21:24 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Biology]]></category>
		<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[Hypertension]]></category>
		<category><![CDATA[Medications]]></category>
		<category><![CDATA[Metabolism]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Neuroplasticity]]></category>
		<category><![CDATA[Neurotransmitters]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[astrocyte]]></category>
		<category><![CDATA[atp]]></category>
		<category><![CDATA[featured]]></category>
		<category><![CDATA[glia]]></category>
		<category><![CDATA[neuroplasticity]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=1201</guid>
		<description><![CDATA[Astrocytes actively participate in activity-dependent synaptic plasticity of neural circuits. Astrocyte activation is both necessary and sufficient for mediating hLTD accompanying LTP induction.  Heterosynaptic long-term depression (hLTD) at synapses accompanying the induction of long-term potentiation (LTP) spatially sharpens the activity-induced synaptic potentiation. Heterosynaptic long-term depression in the hippocampal CA1 region is caused by stimulation-induced ATP release from astrocytes that suppresses transmitter release from synaptic terminals via activation of P2Y receptors. Selective stimulation of astrocytes resulted in LTD of synapses on neighboring neurons. This synaptic modification required Ca(2+) elevation in astrocytes and activation of P2Y receptors, but not N-methyl-D-aspartate receptors.  Blocking P2Y receptors or buffering astrocyte intracellular Ca(2+) at a low level prevented hLTD without affecting LTP]]></description>
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		</item>
		<item>
		<title>Growth Hormone-Releasing Hormone improves Cognition in Older Adults and Adults with  Mild Cognitive Impairment</title>
		<link>http://www.definitivemind.com/2012/10/13/growth-hormone-releasing-hormone-improves-cognition-in-older-adults-and-adults-with-mild-cognitive-impairment/</link>
		<comments>http://www.definitivemind.com/2012/10/13/growth-hormone-releasing-hormone-improves-cognition-in-older-adults-and-adults-with-mild-cognitive-impairment/#comments</comments>
		<pubDate>Sat, 13 Oct 2012 20:35:42 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Cognitive Disorders and Dementia]]></category>
		<category><![CDATA[Endocrinology]]></category>
		<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[Hormones]]></category>
		<category><![CDATA[Medications]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neuroplasticity]]></category>
		<category><![CDATA[Obesity]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[alzheimer's disease]]></category>
		<category><![CDATA[featured]]></category>
		<category><![CDATA[GHRH]]></category>
		<category><![CDATA[growth hormone]]></category>
		<category><![CDATA[growth-hormone releasing-hormone]]></category>
		<category><![CDATA[IGF-1]]></category>
		<category><![CDATA[insulin]]></category>
		<category><![CDATA[mild cognitive impairment]]></category>
		<category><![CDATA[obesity]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=1175</guid>
		<description><![CDATA[Growth hormone-releasing hormone (GHRH), growth hormone, and insulinlike growth factor 1 have potent effects on brain function, their levels decrease with advancing age, and they likely play a role in the pathogenesis of Alzheimer disease. Previously, we reported favorable cognitive effects of short-term GHRH administration in healthy older adults and provided preliminary evidence to suggest a similar benefit in adults with mild cognitive impairment (MCI). The intent-to-treat analysis indicated a favorable effect of GHRH on cognition which was comparable in adults with MCI and healthy older adults. Subsequent analyses indicated a positive GHRH effect on executive function and a trend showing a similar treatment-related benefit in verbal memory. Treatment with GHRH increased insulinlike growth factor 1 levels by 117%, which remained within the physiological range, and reduced percent body fat by 7.4%. Treatment with GHRH increased fasting insulin levels within the normal range by 35% in adults with MCI but not in healthy adults. Adverse events were mild and were reported by 68% of GHRH-treated adults and 36% of those who received placebo.]]></description>
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		<slash:comments>0</slash:comments>
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		<item>
		<title>Thioridazine Inhibits Peroxisomal &#946;-oxidation, Leading to Increased Very Long Chain Fatty Acids and Beta-Amyloid Peptide and an Increased Risk of Alzheimer&#8217;s Disease</title>
		<link>http://www.definitivemind.com/2012/10/10/thioridazine-inhibits-peroxisomal-oxidation-leading-to-increased-very-long-chain-fatty-acids-and-beta-amyloid-peptide-and-an-increased-risk-of-alzheimers-disease/</link>
		<comments>http://www.definitivemind.com/2012/10/10/thioridazine-inhibits-peroxisomal-oxidation-leading-to-increased-very-long-chain-fatty-acids-and-beta-amyloid-peptide-and-an-increased-risk-of-alzheimers-disease/#comments</comments>
		<pubDate>Wed, 10 Oct 2012 09:13:19 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Cognitive Disorders and Dementia]]></category>
		<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychosis]]></category>
		<category><![CDATA[Alzheimer Disease]]></category>
		<category><![CDATA[Antipsychotics]]></category>
		<category><![CDATA[beta-amyloid]]></category>
		<category><![CDATA[beta-oxidation]]></category>
		<category><![CDATA[dementia]]></category>
		<category><![CDATA[fatty acids]]></category>
		<category><![CDATA[featured]]></category>
		<category><![CDATA[thioridazine]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=1169</guid>
		<description><![CDATA[Alzheimer's disease (AD) is characterized by the accumulation of the &#946;-amyloid peptide (A&#946;), which is generated from sequential cleavages of the amyloid precursor protein (APP) by &#946;-secretase (BACE1) and &#947;-secretase. Increased very long chain fatty acid (VLCFA) levels in AD brains imply that peroxisomal &#946;-oxidation dysfunction may be associated with AD pathogenesis. Thioridazine is a selective peroxisomal &#946;-oxidation inhibitor. Thioridazine caused VLCFA accumulation and increases in A&#946;(40) content, APP immunoreactivity and APP(751+770) mRNA expressions in the rat cerebral cortex. Impaired peroxisomal function may play an important role in the progression of AD pathology.]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>A comparison of psychiatry and internal medicine</title>
		<link>http://www.definitivemind.com/2012/09/02/a-comparison-of-psychiatry-and-internal-medicine/</link>
		<comments>http://www.definitivemind.com/2012/09/02/a-comparison-of-psychiatry-and-internal-medicine/#comments</comments>
		<pubDate>Sun, 02 Sep 2012 19:29:14 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[biological psychiatry]]></category>
		<category><![CDATA[featured]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=1042</guid>
		<description><![CDATA[A comparison of psychiatry and internal medicine: a bibliometric study Acad Psychiatry. 2012 Mar 1;36(2):129-32 Stone K, Whitham EA, Ghaemi SN Abstract OBJECTIVE: Psychiatric education needs to expose students to a broad range of topics. One resource for psychiatric education, both during initial training and in later continuing medical education, is the scientific literature, as [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Reduced GABA Concentration in Attention-Deficit/Hyperactivity DisorderReduced GABA Concentration in ADHD.</title>
		<link>http://www.definitivemind.com/2012/09/02/reduced-gaba-concentration-in-attention-deficithyperactivity-disorderreduced-gaba-concentration-in-adhd/</link>
		<comments>http://www.definitivemind.com/2012/09/02/reduced-gaba-concentration-in-attention-deficithyperactivity-disorderreduced-gaba-concentration-in-adhd/#comments</comments>
		<pubDate>Sun, 02 Sep 2012 19:06:33 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Attention Deficit / Hyperactivity Disorder]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Neurotransmitters]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[ADHD]]></category>
		<category><![CDATA[GABA]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=1039</guid>
		<description><![CDATA[Reduced GABA Concentration in Attention-Deficit/Hyperactivity DisorderReduced GABA Concentration in ADHD Arch Gen Psychiatry. 2012 Jul 1;69(7):750-3 Authors: Edden RA, Crocetti D, Zhu H, Gilbert DL, Mostofsky SH Abstract CONTEXT Attention-deficit/hyperactivity disorder (ADHD) is a developmental disorder characterized by a deficit in behavioral inhibition. Recent evidence also suggests a deficit in cortical inhibition via the GABA [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Antidepressant Combination for Major Depression: Flaws in Evidenced-Based Medicine</title>
		<link>http://www.definitivemind.com/2012/08/27/antidepressant-combination-for-major-depression-flaws-in-evidenced-based-medicine/</link>
		<comments>http://www.definitivemind.com/2012/08/27/antidepressant-combination-for-major-depression-flaws-in-evidenced-based-medicine/#comments</comments>
		<pubDate>Mon, 27 Aug 2012 19:14:44 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Depression]]></category>
		<category><![CDATA[Medications]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Study Design Limitations]]></category>
		<category><![CDATA[antidepressants]]></category>
		<category><![CDATA[depression]]></category>
		<category><![CDATA[evidence-based]]></category>
		<category><![CDATA[study design]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=905</guid>
		<description><![CDATA[Antidepressant combination for major depression in incomplete responders-a systematic review. J Affect Disord. 2012 Jul 24; Authors: Lopes Rocha F, Fuzikawa C, Riera R, Ramos MG, Hara C Abstract BACKGROUND: Antidepressant combination has been suggested as a strategy to increase treatment efficacy. The objective of this study was to perform a systematic review and meta-analysis [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Children with ADHD and Anger have a Mood Disorder Too</title>
		<link>http://www.definitivemind.com/2012/08/24/children-with-adhd-with-anger-have-a-mood-disorder-too/</link>
		<comments>http://www.definitivemind.com/2012/08/24/children-with-adhd-with-anger-have-a-mood-disorder-too/#comments</comments>
		<pubDate>Fri, 24 Aug 2012 18:16:28 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[Hypertension]]></category>
		<category><![CDATA[Laboratory Science]]></category>
		<category><![CDATA[Metabolism]]></category>
		<category><![CDATA[Pharmacology]]></category>
		<category><![CDATA[Philosophy]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[RESOURCES]]></category>
		<category><![CDATA[STICKIES]]></category>
		<category><![CDATA[Study Design Limitations]]></category>
		<category><![CDATA[ADHD]]></category>
		<category><![CDATA[bipolar disorder]]></category>
		<category><![CDATA[featured]]></category>
		<category><![CDATA[study design]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=870</guid>
		<description><![CDATA[Pediatric bipolar disorder and ADHD: Family history comparison in the LAMS clinical sample J Affect Disord. 2012 Mar 30; Authors: Arnold LE, Mount K, Frazier T, Demeter C, Youngstrom EA, Fristad MA, Birmaher B, Horwitz S, Findling RL, Kowatch R, Axelson D Abstract &#160; BACKGROUND: Transgenerational association of bipolar spectrum disorder (BPSD) and attention deficit/hyperactivity [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>The Problem of Meta-Analysis of Psychotherapy&#8217;s Effect on Suicidality</title>
		<link>http://www.definitivemind.com/2012/08/24/the-problem-of-meta-analysis-of-psychotherapys-effect-on-suicidality/</link>
		<comments>http://www.definitivemind.com/2012/08/24/the-problem-of-meta-analysis-of-psychotherapys-effect-on-suicidality/#comments</comments>
		<pubDate>Fri, 24 Aug 2012 17:36:50 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Consciousness]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychology]]></category>
		<category><![CDATA[Psychosocial]]></category>
		<category><![CDATA[Psychotherapy]]></category>
		<category><![CDATA[Study Design Limitations]]></category>
		<category><![CDATA[DBT]]></category>
		<category><![CDATA[dialectical behavioral therapy]]></category>
		<category><![CDATA[meta-analysis]]></category>
		<category><![CDATA[psychotherapy]]></category>
		<category><![CDATA[suicidaility]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=867</guid>
		<description><![CDATA[The effects of psychotherapy for adult depression on suicidality and hopelessness: A systematic review and meta-analysis J Affect Disord. 2012 Jul 23; Authors: Cuijpers P, de Beurs DP, van Spijker BA, Berking M, Andersson G, Kerkhof AJ Abstract BACKGROUND: Although treatment guidelines suggest that suicidal patients with depression should be treated for depression with psychotherapy, [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Celebrex is a Potent Antidepressant</title>
		<link>http://www.definitivemind.com/2012/08/23/celecoxib-celebrex-reduces-depression-by-reducing-interleukin-6/</link>
		<comments>http://www.definitivemind.com/2012/08/23/celecoxib-celebrex-reduces-depression-by-reducing-interleukin-6/#comments</comments>
		<pubDate>Fri, 24 Aug 2012 07:46:26 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Cytokines]]></category>
		<category><![CDATA[Eicosinoids]]></category>
		<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Immunology]]></category>
		<category><![CDATA[Mental Health]]></category>
		<category><![CDATA[Mood Disorders]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Pathophysiology]]></category>
		<category><![CDATA[Pharmacology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychology]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[Study Design Limitations]]></category>
		<category><![CDATA[celebrex]]></category>
		<category><![CDATA[celicoxib]]></category>
		<category><![CDATA[depression]]></category>
		<category><![CDATA[IL-6]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=854</guid>
		<description><![CDATA[Effect of celecoxib add-on treatment on symptoms and serum IL-6 concentrations in patients with major depressive disorder: Randomized double-blind placebo-controlled study J Affect Disord. 2012 Apr 18; Authors: Abbasi SH, Hosseini F, Modabbernia A, Ashrafi M, Akhondzadeh S Abstract BACKGROUND: It has been proposed that the mechanism of the antidepressant effect of celecoxib is linked [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Residual Memory Dysfunction In Major Depressive Disorder Means Incomplete Treatment</title>
		<link>http://www.definitivemind.com/2012/08/17/residual-memory-dysfunction-in-recurrent-major-depressive-disorder/</link>
		<comments>http://www.definitivemind.com/2012/08/17/residual-memory-dysfunction-in-recurrent-major-depressive-disorder/#comments</comments>
		<pubDate>Sat, 18 Aug 2012 07:54:15 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Cognitive Disorders and Dementia]]></category>
		<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[Medications]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[depression]]></category>
		<category><![CDATA[memory]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=737</guid>
		<description><![CDATA[Residual memory dysfunction in recurrent major depressive disorder-A longitudinal study from Juntendo University Mood Disorder Project J Affect Disord. 2012 Jul 23; Authors: Maeshima H, Baba H, Nakano Y, Satomura E, Namekawa Y, Takebayashi N, Suzuki T, Mimura M, Arai H Abstract BACKGROUND: Depression may increase the risk of developing Alzheimer&#8217;s disease. Large cohort studies [...]]]></description>
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		</item>
		<item>
		<title>Maternal Gluten Sensitivity Raises Risk for Psychosis in Children</title>
		<link>http://www.definitivemind.com/2012/08/17/maternal-gluten-sensitivity-raises-risk-for-psychosis-in-children/</link>
		<comments>http://www.definitivemind.com/2012/08/17/maternal-gluten-sensitivity-raises-risk-for-psychosis-in-children/#comments</comments>
		<pubDate>Fri, 17 Aug 2012 20:20:38 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Mental Health]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[gluten]]></category>
		<category><![CDATA[psychosis]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=728</guid>
		<description><![CDATA[Maternal antibodies to dietary antigens and risk for nonaffective psychosis in offspring &#160; Am J Psychiatry. 2012 Jun;169(6):625-32 Authors: Karlsson H, Blomstr&#246;m &#197;, Wicks S, Yang S, Yolken RH, Dalman C Abstract OBJECTIVE:The authors analyzed archival dried blood spots obtained from newborns to assess whether levels of immunoglobulin G (IgG) directed at dietary antigens were [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Bereavement Induces Systemic Inflammation</title>
		<link>http://www.definitivemind.com/2012/08/16/bereavement-induced-systemic-inflammation-in-susceptible-persons/</link>
		<comments>http://www.definitivemind.com/2012/08/16/bereavement-induced-systemic-inflammation-in-susceptible-persons/#comments</comments>
		<pubDate>Fri, 17 Aug 2012 00:50:20 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[bereavement]]></category>
		<category><![CDATA[inflammation]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=710</guid>
		<description><![CDATA[When grief makes you sick: Bereavement induced systemic inflammation is a question of genotype Authors: Schultze-Florey CR, Mart&#237;nez-Maza O, Magpantay L, Breen EC, Irwin MR, G&#252;ndel H, O&#8217;Connor MFBrain Behav Immun. 2012 Jun 23; Abstract Although bereavement is associated with increased morbidity and mortality in the surviving spouse, some widow(er)s remain healthy.&#160;Genetic variability in expression [...]]]></description>
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		</item>
		<item>
		<title>Dopamine Dysfunction in Schizophrenia</title>
		<link>http://www.definitivemind.com/2012/08/16/the-nature-of-dopamine-dysfunction-in-schizophrenia/</link>
		<comments>http://www.definitivemind.com/2012/08/16/the-nature-of-dopamine-dysfunction-in-schizophrenia/#comments</comments>
		<pubDate>Thu, 16 Aug 2012 20:23:57 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[dopamine]]></category>
		<category><![CDATA[schizophrenia]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=708</guid>
		<description><![CDATA[The Nature of Dopamine Dysfunction in Schizophrenia and What This Means for Treatment: Meta-analysis of Imaging Studies &#160; Arch Gen Psychiatry. 2012 Apr 2; Authors: Howes OD, Kambeitz J, Kim E, Stahl D, Slifstein M, Abi-Dargham A, Kapur S Abstract CONTEXT: Current drug treatments for schizophrenia are inadequate for many patients, and despite 5 decades [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Eye Movement Tests For Schizophrenia</title>
		<link>http://www.definitivemind.com/2012/08/15/eye-movement-tests-can-distinguish-schizophrenia-cases-from-controls/</link>
		<comments>http://www.definitivemind.com/2012/08/15/eye-movement-tests-can-distinguish-schizophrenia-cases-from-controls/#comments</comments>
		<pubDate>Thu, 16 Aug 2012 07:47:15 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[eye]]></category>
		<category><![CDATA[schizophrenia]]></category>
		<category><![CDATA[test]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=700</guid>
		<description><![CDATA[Simple Viewing Tests Can Detect Eye Movement Abnormalities That Distinguish Schizophrenia Cases from Controls with Exceptional Accuracy. &#160; Biol Psychiatry. 2012 May 22; Authors: Benson PJ, Beedie SA, Shephard E, Giegling I, Rujescu D, St Clair D Abstract BACKGROUND: We have investigated which eye-movement tests alone and combined can best discriminate schizophrenia cases from control [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Because They Are Still Ill&#8230;</title>
		<link>http://www.definitivemind.com/2012/08/14/because-they-are-still-ill/</link>
		<comments>http://www.definitivemind.com/2012/08/14/because-they-are-still-ill/#comments</comments>
		<pubDate>Wed, 15 Aug 2012 07:40:41 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Depression]]></category>
		<category><![CDATA[FEATURED]]></category>
		<category><![CDATA[Pharmacology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Study Design Limitations]]></category>
		<category><![CDATA[depression]]></category>
		<category><![CDATA[rating scales]]></category>
		<category><![CDATA[remission]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=692</guid>
		<description><![CDATA[Why do some depressed outpatients who are in remission according to the Hamilton Depression Rating Scale not consider themselves to be in remission? J Clin Psychiatry. 2012 Jun;73(6):790-5 Authors: Zimmerman M, Martinez JA, Attiullah N, Friedman M, Toba C, Boerescu DA, Rahgeb M Abstract OBJECTIVE: In treatment studies of depression, remission is typically defined narrowly, based [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2012/08/14/because-they-are-still-ill/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Substitute Autism for this finding:  Poor nutrition at age 3 is associated with schizotypal personality at age 23</title>
		<link>http://www.definitivemind.com/2012/08/14/substitue-autism-for-this-finding-poor-nutrition-at-age-3-is-associated-with-schizotypal-personality-at-age-23/</link>
		<comments>http://www.definitivemind.com/2012/08/14/substitue-autism-for-this-finding-poor-nutrition-at-age-3-is-associated-with-schizotypal-personality-at-age-23/#comments</comments>
		<pubDate>Tue, 14 Aug 2012 13:37:23 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[autism]]></category>
		<category><![CDATA[schizotypal]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=672</guid>
		<description><![CDATA[Poor nutrition at age 3 and schizotypal personality at age 23: the mediating role of age 11 cognitive functioning. &#160; Am J Psychiatry. 2012 Aug 1;169(8):822-30 Authors: Venables PH, Raine A Abstract OBJECTIVE:&#160;Poor prenatal nutrition has been associated with schizophrenia spectrum disorders in the Netherlands and China, and&#160;it has been suggested that perinatal and postnatal [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2012/08/14/substitue-autism-for-this-finding-poor-nutrition-at-age-3-is-associated-with-schizotypal-personality-at-age-23/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>N-acetylcysteine reduces Cannabis Dependence</title>
		<link>http://www.definitivemind.com/2012/08/13/n-acetylcysteine-reduces-cannabis-dependence/</link>
		<comments>http://www.definitivemind.com/2012/08/13/n-acetylcysteine-reduces-cannabis-dependence/#comments</comments>
		<pubDate>Tue, 14 Aug 2012 04:39:55 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[marijuana]]></category>
		<category><![CDATA[NAC]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=670</guid>
		<description><![CDATA[A double-blind randomized controlled trial of N-acetylcysteine in cannabis-dependent adolescents. &#160; Am J Psychiatry. 2012 Aug 1;169(8):805-12 Authors: Gray KM, Carpenter MJ, Baker NL, Desantis SM, Kryway E, Hartwell KJ, McRae-Clark AL, Brady KT Abstract OBJECTIVE: Preclinical findings suggest that the over-the-counter supplement N-acetylcysteine (NAC), via glutamate modulation in the nucleus accumbens, holds promise as [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2012/08/13/n-acetylcysteine-reduces-cannabis-dependence/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Metformin reduces antipsychotic-induced amenorrhea and weight gain in women with schizophrenia</title>
		<link>http://www.definitivemind.com/2012/08/13/metformin-reduces-antipsychotic-induced-amenorrhea-and-weight-gain-in-women-with-schizophrenia/</link>
		<comments>http://www.definitivemind.com/2012/08/13/metformin-reduces-antipsychotic-induced-amenorrhea-and-weight-gain-in-women-with-schizophrenia/#comments</comments>
		<pubDate>Tue, 14 Aug 2012 04:29:53 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[amenorrhea]]></category>
		<category><![CDATA[Metformin]]></category>
		<category><![CDATA[weight gain]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=668</guid>
		<description><![CDATA[Metformin for treatment of antipsychotic-induced amenorrhea and weight gain in women with first-episode schizophrenia: a double-blind, randomized, placebo-controlled study &#160; Am J Psychiatry. 2012 Aug 1;169(8):813-21 Authors: Wu RR, Jin H, Gao K, Twamley EW, Ou JJ, Shao P, Wang J, Guo XF, Davis JM, Chan PK, Zhao JP Abstract OBJECTIVE: Data on the treatment [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Glucocorticoids Protect Against Effects of Stress on the Amygdala</title>
		<link>http://www.definitivemind.com/2012/08/12/glucocorticoids-protect-against-effects-of-stress-on-the-amygdala/</link>
		<comments>http://www.definitivemind.com/2012/08/12/glucocorticoids-protect-against-effects-of-stress-on-the-amygdala/#comments</comments>
		<pubDate>Mon, 13 Aug 2012 06:45:19 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[amygdala]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[glucocorticoid]]></category>
		<category><![CDATA[stress]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=664</guid>
		<description><![CDATA[Glucocorticoids Protect Against the Delayed Behavioral and Cellular Effects of Acute Stress on the Amygdala. Biol Psychiatry. 2012 May 7; Authors: Rao RP, Anilkumar S, McEwen BS, Chattarji S Abstract BACKGROUND: A single episode of acute immobilization stress has previously been shown to trigger a delayed onset of anxiety-like behavior and spinogenesis in the basolateral [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2012/08/12/glucocorticoids-protect-against-effects-of-stress-on-the-amygdala/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Antidepressants are still useful &#8211; they increase neurogenesis and angiogenesis</title>
		<link>http://www.definitivemind.com/2012/08/12/antidepressants-are-still-useful-they-increase-neurogenesis-and-angiogenesis/</link>
		<comments>http://www.definitivemind.com/2012/08/12/antidepressants-are-still-useful-they-increase-neurogenesis-and-angiogenesis/#comments</comments>
		<pubDate>Mon, 13 Aug 2012 06:41:03 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[antidepressants]]></category>
		<category><![CDATA[neurogenesis]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=662</guid>
		<description><![CDATA[Hippocampal Angiogenesis and Progenitor Cell Proliferation Are Increased with Antidepressant Use in Major Depression. &#160; Biol Psychiatry. 2012 May 30; Authors: Boldrini M, Hen R, Underwood MD, Rosoklija GB, Dwork AJ, Mann JJ, Arango V Abstract BACKGROUND: Adult neurogenesis is coupled to angiogenesis in neurogenic niches in the&#160;dentate gyrus (DG)&#160;and increased by antidepressants in rodents. [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2012/08/12/antidepressants-are-still-useful-they-increase-neurogenesis-and-angiogenesis/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Low Estrogen Predisposes Women to Posttraumatic Stress Disorder.</title>
		<link>http://www.definitivemind.com/2012/08/12/low-estrogen-predisposes-women-to-posttraumatic-stress-disorder/</link>
		<comments>http://www.definitivemind.com/2012/08/12/low-estrogen-predisposes-women-to-posttraumatic-stress-disorder/#comments</comments>
		<pubDate>Mon, 13 Aug 2012 06:30:07 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Hormones]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[estrogen]]></category>
		<category><![CDATA[PTSD]]></category>
		<category><![CDATA[stress]]></category>
		<category><![CDATA[trauma]]></category>
		<category><![CDATA[women]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=660</guid>
		<description><![CDATA[Estrogen levels are associated with extinction deficits in women with posttraumatic stress disorder. Biol Psychiatry. 2012 Jul 1;72(1):19-24. Epub 2012 Apr 12. Glover EM, Jovanovic T, Mercer KB, Kerley K, Bradley B, Ressler KJ, Norrholm SD. Abstract BACKGROUND:Women are twice as likely to develop posttraumatic stress disorder (PTSD) than men.&#160;As shown in our previous work, [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Shorter Telomeres in Severe Mental Illness</title>
		<link>http://www.definitivemind.com/2012/08/12/shorter-telomeres-in-severe-mental-illness/</link>
		<comments>http://www.definitivemind.com/2012/08/12/shorter-telomeres-in-severe-mental-illness/#comments</comments>
		<pubDate>Mon, 13 Aug 2012 06:13:38 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Metabolism]]></category>
		<category><![CDATA[Mind-Body Medicine]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[mental illness]]></category>
		<category><![CDATA[telomere]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=658</guid>
		<description><![CDATA[Telomeres and Early-Life Stress: An Overview Biol Psychiatry. 2012 Jul 23;&#160; Authors: Price LH, Kao HT, Burgers DE, Carpenter LL, Tyrka AR Abstract The long-term sequelae of adverse early-life experiences have long been a focus in psychiatry, with a historic neurobiological emphasis on physiological systems that are demonstrably stress-responsive, such as the hypothalamic-pituitary-adrenal axis and [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2012/08/12/shorter-telomeres-in-severe-mental-illness/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Dose-Dependent Effects of Endotoxin on Neurobehavioral Functions in Humans</title>
		<link>http://www.definitivemind.com/2012/08/11/dose-dependent-effects-of-endotoxin-on-neurobehavioral-functions-in-humans/</link>
		<comments>http://www.definitivemind.com/2012/08/11/dose-dependent-effects-of-endotoxin-on-neurobehavioral-functions-in-humans/#comments</comments>
		<pubDate>Sat, 11 Aug 2012 16:25:45 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[behavior]]></category>
		<category><![CDATA[endotoxin]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=652</guid>
		<description><![CDATA[Dose-Dependent Effects of Endotoxin on Neurobehavioral Functions in Humans PLoS One. 2011;6(12):e28330. Epub 2011 Dec 2. Grigoleit JS, Kullmann JS, Wolf OT, Hammes F, Wegner A, Jablonowski S, Engler H, Gizewski E, Oberbeck R, Schedlowski M. Abstract Clinical and experimental evidence document that inflammation and increased peripheral cytokine levels are associated with depression-like symptoms and [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2012/08/11/dose-dependent-effects-of-endotoxin-on-neurobehavioral-functions-in-humans/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Microglial Mutation contributes to obsessive-compulsive behavior</title>
		<link>http://www.definitivemind.com/2010/08/01/microglial-mutation-contributes-to-obsessive-compulsive-behavior/</link>
		<comments>http://www.definitivemind.com/2010/08/01/microglial-mutation-contributes-to-obsessive-compulsive-behavior/#comments</comments>
		<pubDate>Mon, 02 Aug 2010 06:47:52 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[microglia]]></category>
		<category><![CDATA[obsessive-compulsive]]></category>
		<category><![CDATA[OCD]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=632</guid>
		<description><![CDATA[&#160; &#65279; Hematopoietic origin of pathological grooming in Hoxb8 mutant mice. Chen SK,&#160;Tvrdik P,&#160;Peden E,&#160;Cho S,&#160;Wu S,&#160;Spangrude G,&#160;Capecchi MR. Cell. 2010 May 28;141(5):775-85. &#160; Mouse Hoxb8 mutants show unexpected behavior manifested by compulsive grooming and hair removal, similar to behavior in humans with the obsessive-compulsive disorder spectrum disorder trichotillomania. &#160; As Hox gene disruption often [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Reducing dietary sodium: the case for caution.</title>
		<link>http://www.definitivemind.com/2010/02/21/reducing-dietary-sodium-the-case-for-caution/</link>
		<comments>http://www.definitivemind.com/2010/02/21/reducing-dietary-sodium-the-case-for-caution/#comments</comments>
		<pubDate>Sun, 21 Feb 2010 09:34:06 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[TOPICS]]></category>
		<category><![CDATA[Vitamins, Minerals and Other Nutrients]]></category>
		<category><![CDATA[salt]]></category>
		<category><![CDATA[sodium]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=573</guid>
		<description><![CDATA[Reducing dietary sodium: the case for caution. JAMA. 2010 Feb 3;303(5):448-9 Authors: Alderman MH QUOTE: Multiple randomized clinical trials (RCTs) have established that reduction of sodium intake sufficient to lower blood pressure also increases sympathetic nerve activity, decreases insulin sensitivity, activates the renin angiotensin system, and stimulates aldosterone secretion. &#8212;&#8212;&#8212;- Low salt diets lead to: [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Low Serum Folate Levels as a Risk Factor for Depression</title>
		<link>http://www.definitivemind.com/2010/02/21/low-serum-folate-levels-as-a-risk-factor-for-depression/</link>
		<comments>http://www.definitivemind.com/2010/02/21/low-serum-folate-levels-as-a-risk-factor-for-depression/#comments</comments>
		<pubDate>Sun, 21 Feb 2010 08:50:34 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Vitamins, Minerals and Other Nutrients]]></category>
		<category><![CDATA[depression]]></category>
		<category><![CDATA[folic acid]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=571</guid>
		<description><![CDATA[Low Serum Folate Levels as a Risk Factor for Depressive Mood in Patients With Chronic Epilepsy J Neuropsychiatry Clin Neurosci 15:64-66, February 2003 Johannes Rösche, Dr.med., M.A., Carmen Uhlmann, Dr.hum.biol., Dipl.Psych. and Walter Fröscher, Prof.Dr.med. This study takes into consideration whether low serum folate levels may contribute to depressive mood in patients with chronic epilepsy. [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Hydrocortisone treatment restores brain metabolism and working memory in PTSD</title>
		<link>http://www.definitivemind.com/2010/02/20/hydrocortisone-treatment-restores-brain-metabolism-and-working-memory-in-ptsd/</link>
		<comments>http://www.definitivemind.com/2010/02/20/hydrocortisone-treatment-restores-brain-metabolism-and-working-memory-in-ptsd/#comments</comments>
		<pubDate>Sun, 21 Feb 2010 07:17:28 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Hormones]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[hydrocortisone]]></category>
		<category><![CDATA[PTSD]]></category>
		<category><![CDATA[stress]]></category>
		<category><![CDATA[trauma]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=569</guid>
		<description><![CDATA[Changes in Relative Glucose Metabolic Rate Following Cortisol Administration in Aging Veterans with Posttraumatic Stress Disorder: An FDG-PET Neuroimaging Study J Neuropsychiatry Clin Neurosci.2009; 21: 132-143 Rachel Yehuda, Ph.D., Philip D. Harvey, Ph.D., Julia A. Golier, M.D., Randall E. Newmark, B.A., Christopher R. Bowie, Ph.D., Janelle J. Wohltmann, B.A., Robert A. Grossman, M.D., James Schmeidler, [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Gut microbes enhance the immune system</title>
		<link>http://www.definitivemind.com/2010/02/07/gut-microbes-enhance-the-immune-system/</link>
		<comments>http://www.definitivemind.com/2010/02/07/gut-microbes-enhance-the-immune-system/#comments</comments>
		<pubDate>Sun, 07 Feb 2010 23:26:00 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[immune system]]></category>
		<category><![CDATA[probiotics]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=565</guid>
		<description><![CDATA[Gut microbes extend reach to systemic innate immunity Nature Medicine 16, 160 (2010). Authors: Dana J Philpott &#038; Stephen E Girardin Microbes in the gut can influence distant events, affecting the function of neutrophils in the circulation of mice (pages 228-231). The findings should lead to new studies examining how intestinal microbes affect immunity. &#8212;&#8212;&#8212;- [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Selenium slows memory decline &#8211; Reducing acetylcholinesterase activity</title>
		<link>http://www.definitivemind.com/2010/02/07/selenium-slows-memory-decline-reducing-acetylcholinesterase-activity/</link>
		<comments>http://www.definitivemind.com/2010/02/07/selenium-slows-memory-decline-reducing-acetylcholinesterase-activity/#comments</comments>
		<pubDate>Sun, 07 Feb 2010 19:52:32 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[acetylcholinesterase]]></category>
		<category><![CDATA[dementia]]></category>
		<category><![CDATA[memory]]></category>
		<category><![CDATA[selenium]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=563</guid>
		<description><![CDATA[Organoselenium improves memory decline in mice: Involvement of acetylcholinesterase activity. Neurosci Lett. 2010 Jan 29; Authors: Pinton S, Rocha JT, Zeni G, Nogueira CW The present study was designed to investigate the possible neuroprotective effect of p,p&#8217;-methoxyl-diphenyl diselenide [(MeOPhSe)(2)] in a model of sporadic dementia of Alzheimer&#8217;s type (SDAT) induced by intracerebroventricular (i.c.v.) injection of [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Selenium prevents cognitive decline and oxidative damage</title>
		<link>http://www.definitivemind.com/2010/02/07/selenium-prevents-cognitive-decline-and-oxidative-damage/</link>
		<comments>http://www.definitivemind.com/2010/02/07/selenium-prevents-cognitive-decline-and-oxidative-damage/#comments</comments>
		<pubDate>Sun, 07 Feb 2010 19:47:06 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[dementia]]></category>
		<category><![CDATA[oxidation]]></category>
		<category><![CDATA[selenium]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=561</guid>
		<description><![CDATA[Selenium prevents cognitive decline and oxidative damage in rat model of streptozotocin-induced experimental dementia of Alzheimer&#8217;s type. Brain Res. 2009 Jul 24;1281:117-27. Epub 2009 Apr 15. Ishrat T, Parveen K, Khan MM, Khuwaja G, Khan MB, Yousuf S, Ahmad A, Shrivastav P, Islam F. Department of Medical Elementology and Toxicology, Neurotoxicology Laboratory, Jamia Hamdard (Hamdard [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Pergolide Reduces Cognitive Deficits Associated with Schizotypal Personality Disorder</title>
		<link>http://www.definitivemind.com/2010/02/06/pergolide-reduces-cognitive-deficits-associated-with-schizotypal-personality-disorder/</link>
		<comments>http://www.definitivemind.com/2010/02/06/pergolide-reduces-cognitive-deficits-associated-with-schizotypal-personality-disorder/#comments</comments>
		<pubDate>Sun, 07 Feb 2010 06:56:59 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[cognitive]]></category>
		<category><![CDATA[deficit]]></category>
		<category><![CDATA[pergolide]]></category>
		<category><![CDATA[personality]]></category>
		<category><![CDATA[schizotypal]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=557</guid>
		<description><![CDATA[Pergolide Treatment of Cognitive Deficits Associated with Schizotypal Personality Disorder: Continued Evidence of the Importance of the Dopamine System in the Schizophrenia Spectrum. Neuropsychopharmacology. 2010 Feb 3; Authors: McClure MM, Harvey PD, Goodman M, Triebwasser J, New A, Koenigsberg HW, Sprung LJ, Flory JD, Siever LJ Cognitive deficits observed in schizophrenia are also frequently found [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Supressing HPA Axis Activity via Exogenous Cortisol Enhances Aggressive Behavior in Females, but not in Males.</title>
		<link>http://www.definitivemind.com/2010/02/06/supressing-adrenal-function-via-exogenous-cortisol-enhances-aggressive-behavior-in-females-but-not-in-males/</link>
		<comments>http://www.definitivemind.com/2010/02/06/supressing-adrenal-function-via-exogenous-cortisol-enhances-aggressive-behavior-in-females-but-not-in-males/#comments</comments>
		<pubDate>Sun, 07 Feb 2010 05:40:38 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[aggression]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[female]]></category>
		<category><![CDATA[HPA Axis]]></category>
		<category><![CDATA[male]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=551</guid>
		<description><![CDATA[Exogenous cortisol enhances aggressive behavior in females, but not in males. Psychoneuroendocrinology. 2010 Feb 1; Böhnke R, Bertsch K, Kruk MR, Richter S, Naumann E The hypothalamic-pituitary-adrenal (HPA) axis plays a major role in the development, elicitation, and enhancement of aggressive behavior in animals. Increasing evidence suggests that this is also true for humans. Here, [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/02/06/supressing-adrenal-function-via-exogenous-cortisol-enhances-aggressive-behavior-in-females-but-not-in-males/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Omega-3 Fatty Acids for Prevention of Psychotic Disorders</title>
		<link>http://www.definitivemind.com/2010/02/06/omega-3-fatty-acids-for-prevention-of-psychotic-disorders/</link>
		<comments>http://www.definitivemind.com/2010/02/06/omega-3-fatty-acids-for-prevention-of-psychotic-disorders/#comments</comments>
		<pubDate>Sat, 06 Feb 2010 19:12:23 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[fatty acids]]></category>
		<category><![CDATA[omega-3]]></category>
		<category><![CDATA[psychosis]]></category>
		<category><![CDATA[schizophrenia]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=549</guid>
		<description><![CDATA[Long-Chain {omega}-3 Fatty Acids for Indicated Prevention of Psychotic Disorders: A Randomized, Placebo-Controlled Trial. Arch Gen Psychiatry. 2010 Feb;67(2):146-54 Amminger GP, Schäfer MR, Papageorgiou K, Klier CM, Cotton SM, Harrigan SM, Mackinnon A, McGorry PD, Berger GE CONTEXT: The use of antipsychotic medication for the prevention of psychotic disorders is controversial. Long-chain omega-3 (omega-3) polyunsaturated [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/02/06/omega-3-fatty-acids-for-prevention-of-psychotic-disorders/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Stress Leads to Long-Term Reduction in Corticosteroid Sensitivity in the Hippocampus</title>
		<link>http://www.definitivemind.com/2010/02/06/stress-leads-to-long-term-reduction-in-corticosteroid-sensitivity-in-the-hippocampus/</link>
		<comments>http://www.definitivemind.com/2010/02/06/stress-leads-to-long-term-reduction-in-corticosteroid-sensitivity-in-the-hippocampus/#comments</comments>
		<pubDate>Sat, 06 Feb 2010 18:40:27 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[hippocampus]]></category>
		<category><![CDATA[stress]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=545</guid>
		<description><![CDATA[Primate Early Life Stress Leads to Long-Term Mild Hippocampal Decreases in Corticosteroid Receptor Expression. Biol Psychiatry. 2010 Feb 2; Arabadzisz D, Diaz-Heitjz R, Knuesel I, Weber E, Pilloud S, Dettling AC, Feldon J, Law AJ, Harrison PJ, Pryce CR BACKGROUND: Expression of mineralocorticoid receptor (MR) and glucocorticoid receptor (GR) genes are moderately reduced in several [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Blueberry Juice May Boost Memory</title>
		<link>http://www.definitivemind.com/2010/01/27/blueberry-juice-may-boost-memory/</link>
		<comments>http://www.definitivemind.com/2010/01/27/blueberry-juice-may-boost-memory/#comments</comments>
		<pubDate>Wed, 27 Jan 2010 14:34:52 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[blueberry]]></category>
		<category><![CDATA[dementia]]></category>
		<category><![CDATA[memory]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=543</guid>
		<description><![CDATA[Blueberry Juice May Boost Memory (Via WebMD Health.) &#8212;&#8212;&#8212;- Blueberry Supplementation Improves Memory in Older Adults J Agric Food Chem. 2010 Jan 4. [Epub ahead of print] Krikorian R, Shidler MD, Nash TA, Kalt W, Vinqvist-Tymchuk MR, Shukitt-Hale B, Joseph JA. The prevalence of dementia is increasing with expansion of the older adult population. In [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Inability to sustain positive emotion in Major Depression reflects failure to maintain Nucleus Accumbens activity</title>
		<link>http://www.definitivemind.com/2010/01/27/inability-to-sustain-positive-emotion-in-major-depression-reflects-failure-to-maintain-nucleus-accumbens-activity/</link>
		<comments>http://www.definitivemind.com/2010/01/27/inability-to-sustain-positive-emotion-in-major-depression-reflects-failure-to-maintain-nucleus-accumbens-activity/#comments</comments>
		<pubDate>Wed, 27 Jan 2010 14:22:53 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[depression]]></category>
		<category><![CDATA[major depression]]></category>
		<category><![CDATA[nucleus accumbens]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=541</guid>
		<description><![CDATA[Reduced capacity to sustain positive emotion in major depression reflects diminished maintenance of fronto-striatal brain activation.: &#8220; Proc Natl Acad Sci U S A. 2009 Dec 29;106(52):22445-50 Authors: Heller AS, Johnstone T, Shackman AJ, Light SN, Peterson MJ, Kolden GG, Kalin NH, Davidson RJ Anhedonia, the loss of pleasure or interest in previously rewarding stimuli, [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Brain Scan for Post-Traumatic Stress Disorder</title>
		<link>http://www.definitivemind.com/2010/01/27/brain-scan-for-post-traumatic-stress-disorder/</link>
		<comments>http://www.definitivemind.com/2010/01/27/brain-scan-for-post-traumatic-stress-disorder/#comments</comments>
		<pubDate>Wed, 27 Jan 2010 14:15:42 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[brain]]></category>
		<category><![CDATA[PTSD]]></category>
		<category><![CDATA[scan]]></category>
		<category><![CDATA[stress]]></category>
		<category><![CDATA[trauma]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=537</guid>
		<description><![CDATA[Brain Scan Offers First Biological Test in Diagnosis of Post-Traumatic Stress Disorder Scientific American &#8211; Psychiatry 1/22/10 Carina Storrs The synchronous neural interactions test as a functional neuromarker for post-traumatic stress disorder (PTSD): a robust classification method based on the bootstrap. J Neural Eng. 2010 Feb;7(1):16011. Epub 2010 Jan 20. Georgopoulos AP, Tan HR, Lewis [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Reduction in CRF Receptor type 1 expression in the Amygdala reduces Anxiety</title>
		<link>http://www.definitivemind.com/2010/01/22/the-anxiolytic-effect-of-environmental-enrichment-is-mediated-via-amygdalar-crf-receptor-type-1/</link>
		<comments>http://www.definitivemind.com/2010/01/22/the-anxiolytic-effect-of-environmental-enrichment-is-mediated-via-amygdalar-crf-receptor-type-1/#comments</comments>
		<pubDate>Fri, 22 Jan 2010 17:53:30 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[amygdala]]></category>
		<category><![CDATA[anxiety]]></category>
		<category><![CDATA[corticotropin]]></category>
		<category><![CDATA[CRF]]></category>
		<category><![CDATA[CRH]]></category>
		<category><![CDATA[hormone]]></category>
		<category><![CDATA[releasing]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=531</guid>
		<description><![CDATA[The anxiolytic effect of environmental enrichment is mediated via amygdalar CRF receptor type 1. Mol Psychiatry. 2010 Jan 19; Authors: Sztainberg Y, Kuperman Y, Tsoory M, Lebow M, Chen A Environmental enrichment (EE) is known to have an anxiolytic effect in several animal models; however, the molecular mechanisms underlying these behavioral changes are not understood. [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/22/the-anxiolytic-effect-of-environmental-enrichment-is-mediated-via-amygdalar-crf-receptor-type-1/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>CRF stimulates Pancreatic Beta Cell Insulin Secretion and Beta Cell Proliferation</title>
		<link>http://www.definitivemind.com/2010/01/22/crf-stimulates-pancreatic-beta-cell-insulin-secretion-and-beta-cell-proliferation/</link>
		<comments>http://www.definitivemind.com/2010/01/22/crf-stimulates-pancreatic-beta-cell-insulin-secretion-and-beta-cell-proliferation/#comments</comments>
		<pubDate>Fri, 22 Jan 2010 17:50:03 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[beta cell]]></category>
		<category><![CDATA[corticotropin]]></category>
		<category><![CDATA[CRF]]></category>
		<category><![CDATA[CRH]]></category>
		<category><![CDATA[hormone]]></category>
		<category><![CDATA[insulin]]></category>
		<category><![CDATA[pancreas]]></category>
		<category><![CDATA[releasing]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=527</guid>
		<description><![CDATA[CRFR1 is expressed on pancreatic beta cells, promotes beta cell proliferation, and potentiates insulin secretion in a glucose-dependent manner. Proc Natl Acad Sci U S A. 2010 Jan 12;107(2):912-7 Authors: Huising MO, van der Meulen T, Vaughan JM, Matsumoto M, Donaldson CJ, Park H, Billestrup N, Vale WW Corticotropin-releasing factor (CRF), originally characterized as the [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Levetiracetam inhibits interleukin-1beta inflammatory responses in the hippocampus and piriform cortex</title>
		<link>http://www.definitivemind.com/2010/01/20/levetiracetam-inhibits-interleukin-1beta-inflammatory-responses-in-the-hippocampus-and-piriform-cortex/</link>
		<comments>http://www.definitivemind.com/2010/01/20/levetiracetam-inhibits-interleukin-1beta-inflammatory-responses-in-the-hippocampus-and-piriform-cortex/#comments</comments>
		<pubDate>Thu, 21 Jan 2010 07:51:23 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Medications]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[hippocampus]]></category>
		<category><![CDATA[IL-1b]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[interleukin]]></category>
		<category><![CDATA[levetiracetam]]></category>
		<category><![CDATA[piriform cortex]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=525</guid>
		<description><![CDATA[Levetiracetam inhibits interleukin-1beta inflammatory responses in the hippocampus and piriform cortex of epileptic rats. Neurosci Lett. 2010 Jan 14; Authors: Kim JE, Choi HC, Song HK, Jo SM, Kim DS, Choi SY, Kim YI, Kang TC Levetiracetam (LEV, 2S-(oxo-1-pyrrolidinyl)butanamide, Keppra(R), UCB Pharma) is a new antiepileptic drug used to treat certain types of seizures in [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/20/levetiracetam-inhibits-interleukin-1beta-inflammatory-responses-in-the-hippocampus-and-piriform-cortex/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>BDNF variant from Val66Met polymorphism reduces HPA-axis reactivity to acute stress.</title>
		<link>http://www.definitivemind.com/2010/01/19/bdnf-variant-from-val66met-polymorphism-reduces-hpa-axis-reactivity-to-acute-stress/</link>
		<comments>http://www.definitivemind.com/2010/01/19/bdnf-variant-from-val66met-polymorphism-reduces-hpa-axis-reactivity-to-acute-stress/#comments</comments>
		<pubDate>Wed, 20 Jan 2010 06:53:49 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[BDNF]]></category>
		<category><![CDATA[HPA Axis]]></category>
		<category><![CDATA[stress]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=515</guid>
		<description><![CDATA[The BDNF Val66Met polymorphism affects HPA-axis reactivity to acute stress. Psychoneuroendocrinology. 2010 Jan 13; Authors: Alexander N, Osinsky R, Schmitz A, Mueller E, Kuepper Y, Hennig J BACKGROUND: Growing evidence suggests that individual differences in HPA-axis reactivity to psychosocial stress are partly due to heritable influences. However, knowledge about the role of specific genetic variants [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>The major mood disorders have significant genetic overlap</title>
		<link>http://www.definitivemind.com/2010/01/19/the-major-mood-disorders-have-significant-genetic-overlap/</link>
		<comments>http://www.definitivemind.com/2010/01/19/the-major-mood-disorders-have-significant-genetic-overlap/#comments</comments>
		<pubDate>Tue, 19 Jan 2010 21:05:07 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[genetic]]></category>
		<category><![CDATA[mood]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=513</guid>
		<description><![CDATA[Meta-analysis of genome-wide association data identifies a risk locus for major mood disorders on 3p21.1 Nature Genetics Published online: 17 January 2010 &#124; doi:10.1038/ng.523 Francis J McMahon1, Nirmala Akula1, Thomas G Schulze1,2, Pierandrea Muglia3,4, Federica Tozzi3, Sevilla D Detera-Wadleigh1, C J M Steele1, René Breuer2, Jana Strohmaier2, Jens R Wendland1, Manuel Mattheisen5,6,7, Thomas W Mühleisen5,6, [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Methylprednisolone reduces nitric oxide formation in cytokine-stimulated oligodendroglial cells</title>
		<link>http://www.definitivemind.com/2010/01/18/methylprednisolone-reduces-nitric-oxide-formation-in-cytokine-stimulated-oligodendroglial-cells/</link>
		<comments>http://www.definitivemind.com/2010/01/18/methylprednisolone-reduces-nitric-oxide-formation-in-cytokine-stimulated-oligodendroglial-cells/#comments</comments>
		<pubDate>Tue, 19 Jan 2010 01:20:31 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[cytokines]]></category>
		<category><![CDATA[glia]]></category>
		<category><![CDATA[medrol]]></category>
		<category><![CDATA[methylprednisolone]]></category>
		<category><![CDATA[nitric oxide]]></category>
		<category><![CDATA[oligodendrocyte]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=509</guid>
		<description><![CDATA[Effects of methylprednisolone and glatiramer acetate on nitric oxide formation of cytokine-stimulated cells from the rat oligodendroglial cell line OLN-93. Neuroimmunomodulation. 2010;17(1):23-30 Authors: Holzknecht C, Röhl C OBJECTIVES: In multiple sclerosis, an autoimmune inflammatory disease, oligodendroglia are primarily affected and play an important role in the onset and process of the degeneration of neuronal axons. [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/18/methylprednisolone-reduces-nitric-oxide-formation-in-cytokine-stimulated-oligodendroglial-cells/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
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		<title>Activated microglia increase excitability of rat hippocampal neurons by producing interleukin-1beta.</title>
		<link>http://www.definitivemind.com/2010/01/18/activated-microglia-increase-excitability-of-rat-hippocampal-neurons-by-producing-interleukin-1beta/</link>
		<comments>http://www.definitivemind.com/2010/01/18/activated-microglia-increase-excitability-of-rat-hippocampal-neurons-by-producing-interleukin-1beta/#comments</comments>
		<pubDate>Tue, 19 Jan 2010 01:06:31 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[activated]]></category>
		<category><![CDATA[hippocampus]]></category>
		<category><![CDATA[IL-1b]]></category>
		<category><![CDATA[interleukin]]></category>
		<category><![CDATA[microglia]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=507</guid>
		<description><![CDATA[Kainic acid-activated microglia mediate increased excitability of rat hippocampal neurons in vitro and in vivo: crucial role of interleukin-1beta. Neuroimmunomodulation. 2010;17(1):31-8 Authors: Zheng H, Zhu W, Zhao H, Wang X, Wang W, Li Z OBJECTIVE: It is known that highly activated microglia and the consequent production of inflammatory cytokines were associated with neuroexcitotoxic injuries. The [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/18/activated-microglia-increase-excitability-of-rat-hippocampal-neurons-by-producing-interleukin-1beta/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Inflammatory biomarkers in patients with posttraumatic stress disorder</title>
		<link>http://www.definitivemind.com/2010/01/18/inflammatory-biomarkers-in-patients-with-posttraumatic-stress-disorder/</link>
		<comments>http://www.definitivemind.com/2010/01/18/inflammatory-biomarkers-in-patients-with-posttraumatic-stress-disorder/#comments</comments>
		<pubDate>Tue, 19 Jan 2010 01:00:11 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[PTSD]]></category>
		<category><![CDATA[stress]]></category>
		<category><![CDATA[trauma]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=503</guid>
		<description><![CDATA[Inflammatory biomarkers in patients with posttraumatic stress disorder caused by myocardial infarction and the role of depressive symptoms.: &#8220; Neuroimmunomodulation. 2010;17(1):39-46 Authors: von Känel R, Begré S, Abbas CC, Saner H, Gander ML, Schmid JP OBJECTIVE: Inflammation might link posttraumatic stress disorder (PTSD) with an increased risk of cardiovascular events. We explored the association between [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Increased Pro-Inflammatory Activity underlying Posttraumatic Stress Disorder</title>
		<link>http://www.definitivemind.com/2010/01/18/increased-pro-inflammatory-activity-underlying-posttraumatic-stress-disorder/</link>
		<comments>http://www.definitivemind.com/2010/01/18/increased-pro-inflammatory-activity-underlying-posttraumatic-stress-disorder/#comments</comments>
		<pubDate>Tue, 19 Jan 2010 00:56:31 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[PTSD]]></category>
		<category><![CDATA[stress]]></category>
		<category><![CDATA[trauma]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=499</guid>
		<description><![CDATA[Evidence for low-grade systemic proinflammatory activity in patients with posttraumatic stress disorder. J Psychiatr Res. 2007 Nov;41(9):744-52. Epub 2006 Aug 9. von Känel R, Hepp U, Kraemer B, Traber R, Keel M, Mica L, Schnyder U. Department of General Internal Medicine, Division of Psychosomatic Medicine, University Hospital/INSELSPITAL, Freiburgstrasse 4, CH-3010 Berne, Switzerland. roland.vonkaenel@insel.ch Posttraumatic stress [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/18/increased-pro-inflammatory-activity-underlying-posttraumatic-stress-disorder/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Pro-inflammatory cytokine activation of the hypothalamic-pituitary-adrenal axis via NF-kappaB and Nurr-1 expression</title>
		<link>http://www.definitivemind.com/2010/01/18/pro-inflammatory-cytokine-activation-of-the-hypothalamic-pituitary-adrenal-axis-via-nf-kappab-and-nurr-1-expression/</link>
		<comments>http://www.definitivemind.com/2010/01/18/pro-inflammatory-cytokine-activation-of-the-hypothalamic-pituitary-adrenal-axis-via-nf-kappab-and-nurr-1-expression/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 23:55:07 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cytokine]]></category>
		<category><![CDATA[HPA Axis]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[NF-kappB]]></category>
		<category><![CDATA[Nurr-1]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=495</guid>
		<description><![CDATA[Involvement of Nuclear Factor-kappaB and Nurr-1 in Cytokine-Induced Transcription of Proopiomelanocortin Gene in AtT20 Corticotroph Cells. Neuroimmunomodulation. 2009 Nov 17;17(2):88-96 Authors: Takayasu S, Iwasaki Y, Nigawara T, Asai M, Yoshida M, Kageyama K, Suda T Objective: The precise mechanism whereby proinflammatory cytokines activate the hypothalamo-pituitary-adrenal axis is still unclear. We examined whether transcription factors nuclear [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Testosterone improves Muscle Strength, Physical Function, Body Composition, and Quality of Life in Intermediate-Frail and Frail Elderly Men</title>
		<link>http://www.definitivemind.com/2010/01/18/testosterone-improves-muscle-strength-physical-function-body-composition-and-quality-of-life-in-intermediate-frail-and-frail-elderly-men/</link>
		<comments>http://www.definitivemind.com/2010/01/18/testosterone-improves-muscle-strength-physical-function-body-composition-and-quality-of-life-in-intermediate-frail-and-frail-elderly-men/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 23:34:22 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Hormones]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[body composition]]></category>
		<category><![CDATA[elderly]]></category>
		<category><![CDATA[male]]></category>
		<category><![CDATA[muscle strength]]></category>
		<category><![CDATA[quality of life]]></category>
		<category><![CDATA[testosterone]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=491</guid>
		<description><![CDATA[Effects of Testosterone on Muscle Strength, Physical Function, Body Composition, and Quality of Life in Intermediate-Frail and Frail Elderly Men: A Randomized, Double-Blind, Placebo-Controlled Study. J Clin Endocrinol Metab. 2010 Jan 8; Authors: Srinivas-Shankar U, Roberts SA, Connolly MJ, O&#8217; Connell MD, Adams JE, Oldham JA, Wu FC Context: Physical frailty is associated with reduced [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Thiazolidinedione associated with increased Risk of Fractures in Patients with Type 2 Diabetes Mellitus.</title>
		<link>http://www.definitivemind.com/2010/01/18/thiazolidinedione-associated-with-increased-risk-of-fractures-in-patients-with-type-2-diabetes-mellitus/</link>
		<comments>http://www.definitivemind.com/2010/01/18/thiazolidinedione-associated-with-increased-risk-of-fractures-in-patients-with-type-2-diabetes-mellitus/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 23:23:52 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Medications]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[diabetes]]></category>
		<category><![CDATA[fracture]]></category>
		<category><![CDATA[thiazolidinedione]]></category>
		<category><![CDATA[type 2]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=489</guid>
		<description><![CDATA[Thiazolidinedione Use and the Longitudinal Risk of Fractures in Patients with Type 2 Diabetes Mellitus. J Clin Endocrinol Metab. 2010 Jan 8; Authors: Habib ZA, Havstad SL, Wells K, Divine G, Pladevall M, Williams LK Context: Thiazolidinedione (TZD) use has recently been associated with an increased risk of fractures. Objective: The aim of this study [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Long-Term Growth Hormone Therapy Improves Body Composition and Motor Function</title>
		<link>http://www.definitivemind.com/2010/01/18/long-term-growth-hormone-therapy-improves-body-composition-and-motor-function/</link>
		<comments>http://www.definitivemind.com/2010/01/18/long-term-growth-hormone-therapy-improves-body-composition-and-motor-function/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 23:16:55 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[body composition]]></category>
		<category><![CDATA[growth hormone]]></category>
		<category><![CDATA[motor function]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=485</guid>
		<description><![CDATA[Long-Term Growth Hormone Therapy Changes the Natural History of Body Composition and Motor Function in Children with Prader-Willi Syndrome. J Clin Endocrinol Metab. 2010 Jan 8; Authors: Carrel AL, Myers SE, Whitman BY, Eickhoff J, Allen DB Background: Children with Prader-Willi syndrome (PWS) have decreased muscle mass, hypotonia, and impaired linear growth. Recombinant human GH [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Low Total Testosterone increases risk for Abdominal Aortic Aneurysm</title>
		<link>http://www.definitivemind.com/2010/01/18/low-total-testosterone-increases-risk-for-abdominal-aortic-aneurysm/</link>
		<comments>http://www.definitivemind.com/2010/01/18/low-total-testosterone-increases-risk-for-abdominal-aortic-aneurysm/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 23:09:57 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[aortic aneurysm]]></category>
		<category><![CDATA[testosterone]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=483</guid>
		<description><![CDATA[Associations of Total Testosterone, Sex Hormone-Binding Globulin, Calculated Free Testosterone, and Luteinizing Hormone with Prevalence of Abdominal Aortic Aneurysm in Older Men. J Clin Endocrinol Metab. 2010 Jan 8; Authors: Yeap BB, Hyde Z, Norman PE, Chubb SA, Golledge J Context: Abdominal aortic aneurysm (AAA) is associated with mortality in older adults, and increasing aortic [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Leptin Prevents Weight Loss-Induced Slowing of Metabolism</title>
		<link>http://www.definitivemind.com/2010/01/18/leptin-prevents-weight-loss-induced-slowing-of-metabolism/</link>
		<comments>http://www.definitivemind.com/2010/01/18/leptin-prevents-weight-loss-induced-slowing-of-metabolism/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 23:06:37 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[leptin]]></category>
		<category><![CDATA[metabolism]]></category>
		<category><![CDATA[obesity]]></category>
		<category><![CDATA[weight loss]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=479</guid>
		<description><![CDATA[Leptin Replacement Prevents Weight Loss-Induced Metabolic Adaptation in Congenital Leptin-Deficient Patients. J Clin Endocrinol Metab. 2010 Jan 8; Authors: Galgani JE, Greenway FL, Caglayan S, Wong ML, Licinio J, Ravussin E Context: Leptin regulates energy homeostasis by suppressing food intake; however, its role in energy expenditure and fat oxidation remains uncertain in humans. Objective: The [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Obesity with Insulin Resistance Is Associated with a Proinflammatory and Prothrombotic State</title>
		<link>http://www.definitivemind.com/2010/01/18/obesity-with-insulin-resistance-is-associated-with-a-proinflammatory-and-prothrombotic-state/</link>
		<comments>http://www.definitivemind.com/2010/01/18/obesity-with-insulin-resistance-is-associated-with-a-proinflammatory-and-prothrombotic-state/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 22:57:08 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[insulin]]></category>
		<category><![CDATA[obesity]]></category>
		<category><![CDATA[resistance]]></category>
		<category><![CDATA[thrombosis]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=469</guid>
		<description><![CDATA[Obesity without Established Comorbidities of the Metabolic Syndrome Is Associated with a Proinflammatory and Prothrombotic State, Even before the Onset of Puberty in Children. J Clin Endocrinol Metab. 2010 Jan 8; Authors: Mauras N, Delgiorno C, Kollman C, Bird K, Morgan M, Sweeten S, Balagopal P, Damaso L Background: Metabolic syndrome (MS)-related comorbidities in obesity, [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Regional differences in grey matter in bipolar disorder and schizophrenia</title>
		<link>http://www.definitivemind.com/2010/01/18/regional-differences-in-grey-matter-in-bipolar-disorder-and-schizophrenia/</link>
		<comments>http://www.definitivemind.com/2010/01/18/regional-differences-in-grey-matter-in-bipolar-disorder-and-schizophrenia/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 18:20:40 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[bipolar]]></category>
		<category><![CDATA[gray matter]]></category>
		<category><![CDATA[schizophrenia]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=465</guid>
		<description><![CDATA[Anatomy of bipolar disorder and schizophrenia: A meta-analysis. Schizophr Res. 2010 Jan 11; Authors: Ellison-Wright I, Bullmore E BACKGROUND: Recent genetic results have indicated that the two major, classically distinct forms of psychosis &#8211; schizophrenia and bipolar disorder &#8211; may share causative factors in common. However it is not clear to what extent they may [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Emotions and Hallucinations &#8211; Increased amygdala and parahippocampal gyrus activation in schizophrenic patients with auditory hallucinations</title>
		<link>http://www.definitivemind.com/2010/01/18/emotions-and-hallucinations-increased-amygdala-and-parahippocampal-gyrus-activation-in-schizophrenic-patients-with-auditory-hallucinations/</link>
		<comments>http://www.definitivemind.com/2010/01/18/emotions-and-hallucinations-increased-amygdala-and-parahippocampal-gyrus-activation-in-schizophrenic-patients-with-auditory-hallucinations/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 18:14:57 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[amygdala]]></category>
		<category><![CDATA[auditory]]></category>
		<category><![CDATA[emotion]]></category>
		<category><![CDATA[hallucination]]></category>
		<category><![CDATA[parahippocampal gyrus]]></category>
		<category><![CDATA[schizophrenia]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=463</guid>
		<description><![CDATA[Increased amygdala and parahippocampal gyrus activation in schizophrenic patients with auditory hallucinations: An fMRI study using independent component analysis. Schizophr Res. 2010 Jan 11; Authors: Escartí MJ, de la Iglesia-Vayá M, Martí-Bonmatí L, Robles M, Carbonell J, Lull JJ, García-Martí G, Manjón JV, Aguilar EJ, Aleman A, Sanjuán J OBJECTIVE: Hallucinations in patients with schizophrenia [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/18/emotions-and-hallucinations-increased-amygdala-and-parahippocampal-gyrus-activation-in-schizophrenic-patients-with-auditory-hallucinations/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Stress, Inflammation, and Yoga Practice.</title>
		<link>http://www.definitivemind.com/2010/01/17/stress-inflammation-and-yoga-practice/</link>
		<comments>http://www.definitivemind.com/2010/01/17/stress-inflammation-and-yoga-practice/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 06:59:16 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Exercise]]></category>
		<category><![CDATA[Mind-Body Medicine]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[stress]]></category>
		<category><![CDATA[yoga]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=457</guid>
		<description><![CDATA[Stress, Inflammation, and Yoga Practice.: &#8220; Psychosom Med. 2010 Jan 11; Authors: Kiecolt-Glaser JK, Christian L, Preston H, Houts CR, Malarkey WB, Emery CF, Glaser R Objective: To address the mechanisms underlying hatha yoga&#8217;s potential stress-reduction benefits, we compared inflammatory and endocrine responses of novice and expert yoga practitioners before, during, and after a restorative [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/17/stress-inflammation-and-yoga-practice/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Pyrogenic cytokines did not mediate a stress interview-induced hyperthermic response in a patient with psychogenic fever: a case report.</title>
		<link>http://www.definitivemind.com/2010/01/17/pyrogenic-cytokines-did-not-mediate-a-stress-interview-induced-hyperthermic-response-in-a-patient-with-psychogenic-fever-a-case-report/</link>
		<comments>http://www.definitivemind.com/2010/01/17/pyrogenic-cytokines-did-not-mediate-a-stress-interview-induced-hyperthermic-response-in-a-patient-with-psychogenic-fever-a-case-report/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 06:50:47 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cytokine]]></category>
		<category><![CDATA[fever]]></category>
		<category><![CDATA[hyperthermia]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[proinflammatory]]></category>
		<category><![CDATA[stress]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=455</guid>
		<description><![CDATA[Pyrogenic cytokines did not mediate a stress interview-induced hyperthermic response in a patient with psychogenic fever: a case report. Psychosom Med. 2009 Nov;71(9):932-6 Authors: Hiramoto T, Oka T, Yoshihara K, Kubo C OBJECTIVE: To investigate if pyrogenic cytokines mediated psychological stress-induced hyperthermic response in a patient with psychogenic fever. Despite many case reports on psychogenic [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/17/pyrogenic-cytokines-did-not-mediate-a-stress-interview-induced-hyperthermic-response-in-a-patient-with-psychogenic-fever-a-case-report/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Social stress desensitizes lymphocytes to regulation by endogenous glucocorticoids</title>
		<link>http://www.definitivemind.com/2010/01/17/social-stress-desensitizes-lymphocytes-to-regulation-by-endogenous-glucocorticoids/</link>
		<comments>http://www.definitivemind.com/2010/01/17/social-stress-desensitizes-lymphocytes-to-regulation-by-endogenous-glucocorticoids/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 06:37:35 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[glucocorticoid]]></category>
		<category><![CDATA[lymphocytes]]></category>
		<category><![CDATA[social]]></category>
		<category><![CDATA[stress]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=453</guid>
		<description><![CDATA[Social stress desensitizes lymphocytes to regulation by endogenous glucocorticoids: insights from in vivo cell trafficking dynamics in rhesus macaques. Psychosom Med. 2009 Jul;71(6):591-7 Authors: Cole SW, Mendoza SP, Capitanio JP OBJECTIVE: To determine whether chronic social stress can desensitize leukocytes to normal physiologic regulation by endogenous glucocorticoids. METHODS: We analyzed the longitudinal relationship between plasma [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/17/social-stress-desensitizes-lymphocytes-to-regulation-by-endogenous-glucocorticoids/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
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		<title>Decreased gray matter volumes in the cingulo-frontal cortex and the amygdala in patients with fibromyalgia.</title>
		<link>http://www.definitivemind.com/2010/01/17/decreased-gray-matter-volumes-in-the-cingulo-frontal-cortex-and-the-amygdala-in-patients-with-fibromyalgia/</link>
		<comments>http://www.definitivemind.com/2010/01/17/decreased-gray-matter-volumes-in-the-cingulo-frontal-cortex-and-the-amygdala-in-patients-with-fibromyalgia/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 06:26:39 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[amygdala]]></category>
		<category><![CDATA[cingular]]></category>
		<category><![CDATA[fibromyalgia]]></category>
		<category><![CDATA[frontal cortex]]></category>
		<category><![CDATA[gray matter]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=451</guid>
		<description><![CDATA[Decreased gray matter volumes in the cingulo-frontal cortex and the amygdala in patients with fibromyalgia. Psychosom Med. 2009 Jun;71(5):566-73 Authors: Burgmer M, Gaubitz M, Konrad C, Wrenger M, Hilgart S, Heuft G, Pfleiderer B OBJECTIVE: Studies in fibromyalgia syndrome with functional neuroimaging support the hypothesis of central pain augmentation. To determine whether structural changes in [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/17/decreased-gray-matter-volumes-in-the-cingulo-frontal-cortex-and-the-amygdala-in-patients-with-fibromyalgia/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
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		<title>Cytokine polymorphisms in the pathophysiology of mood disorders.</title>
		<link>http://www.definitivemind.com/2010/01/17/cytokine-polymorphisms-in-the-pathophysiology-of-mood-disorders/</link>
		<comments>http://www.definitivemind.com/2010/01/17/cytokine-polymorphisms-in-the-pathophysiology-of-mood-disorders/#comments</comments>
		<pubDate>Mon, 18 Jan 2010 01:56:35 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cytokine]]></category>
		<category><![CDATA[depression]]></category>
		<category><![CDATA[mood]]></category>
		<category><![CDATA[pathophysiology]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=447</guid>
		<description><![CDATA[Cytokine polymorphisms in the pathophysiology of mood disorders. CNS Spectr. 2009 Aug;14(8):419-25 Authors: Clerici M, Arosio B, Mundo E, Cattaneo E, Pozzoli S, Dell&#8217;osso B, Vergani C, Trabattoni D, Altamura AC INTRODUCTION: An increasing amount of data suggests that dysregulation of the immune system, including the cytokine network, is associated with the etiology and pathophysiology [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/17/cytokine-polymorphisms-in-the-pathophysiology-of-mood-disorders/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Omega-3 Fatty Acids improve macro- and microvascular function in subjects with type 2 diabetes mellitus.</title>
		<link>http://www.definitivemind.com/2010/01/17/omega-3-fatty-acids-improve-macro-and-microvascular-function-in-subjects-with-type-2-diabetes-mellitus/</link>
		<comments>http://www.definitivemind.com/2010/01/17/omega-3-fatty-acids-improve-macro-and-microvascular-function-in-subjects-with-type-2-diabetes-mellitus/#comments</comments>
		<pubDate>Sun, 17 Jan 2010 21:28:42 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[diabetes]]></category>
		<category><![CDATA[fatty acid]]></category>
		<category><![CDATA[omega-3]]></category>
		<category><![CDATA[type 2]]></category>
		<category><![CDATA[vascular function]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=445</guid>
		<description><![CDATA[Effects of n-3 fatty acids on macro- and microvascular function in subjects with type 2 diabetes mellitus. Am J Clin Nutr. 2010 Jan 13; Authors: Stirban A, Nandrean S, Götting C, Tamler R, Pop A, Negrean M, Gawlowski T, Stratmann B, Tschoepe D BACKGROUND: Recent evidence supports the protective effects of n-3 (omega-3) fatty acids [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/17/omega-3-fatty-acids-improve-macro-and-microvascular-function-in-subjects-with-type-2-diabetes-mellitus/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Testosterone for Migraine</title>
		<link>http://www.definitivemind.com/2010/01/16/testosterone-for-migraine/</link>
		<comments>http://www.definitivemind.com/2010/01/16/testosterone-for-migraine/#comments</comments>
		<pubDate>Sun, 17 Jan 2010 02:10:38 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Hormones]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[migraine]]></category>
		<category><![CDATA[testosterone]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=443</guid>
		<description><![CDATA[Androgenic suppression of spreading depression in familial hemiplegic migraine type 1 mutant mice. Ann Neurol. 2009 Oct;66(4):564-8 Authors: Eikermann-Haerter K, Baum MJ, Ferrari MD, van den Maagdenberg AM, Moskowitz MA, Ayata C Familial hemiplegic migraine type 1 (FHM1), a severe migraine with aura variant, is caused by mutations in the CACNA1A gene. Mutant mice carrying [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/16/testosterone-for-migraine/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Role of endogenous testosterone concentration in pediatric stroke.</title>
		<link>http://www.definitivemind.com/2010/01/16/role-of-endogenous-testosterone-concentration-in-pediatric-stroke/</link>
		<comments>http://www.definitivemind.com/2010/01/16/role-of-endogenous-testosterone-concentration-in-pediatric-stroke/#comments</comments>
		<pubDate>Sun, 17 Jan 2010 01:30:30 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[General Medicine]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[stroke]]></category>
		<category><![CDATA[testosterone]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=441</guid>
		<description><![CDATA[Role of endogenous testosterone concentration in pediatric stroke. Ann Neurol. 2009 Dec;66(6):754-8 Authors: Normann S, de Veber G, Fobker M, Langer C, Kenet G, Bernard TJ, Fiedler B, Sträter R, Goldenberg NA, Nowak-Göttl U Previous studies have indicated a male predominance in pediatric stroke. To elucidate this gender disparity, total testosterone concentration was measured in [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/16/role-of-endogenous-testosterone-concentration-in-pediatric-stroke/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>The Subthalamic Nucleus and Impulsivity</title>
		<link>http://www.definitivemind.com/2010/01/16/the-subthalamic-nucleus-and-impulsivity/</link>
		<comments>http://www.definitivemind.com/2010/01/16/the-subthalamic-nucleus-and-impulsivity/#comments</comments>
		<pubDate>Sun, 17 Jan 2010 01:22:26 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[impulsivity]]></category>
		<category><![CDATA[subthalamic nucleus]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=439</guid>
		<description><![CDATA[Stimulation of the subthalamic nucleus and impulsivity: release your horses. Ann Neurol. 2009 Dec;66(6):817-24 Authors: Ballanger B, van Eimeren T, Moro E, Lozano AM, Hamani C, Boulinguez P, Pellecchia G, Houle S, Poon YY, Lang AE, Strafella AP OBJECTIVE: In Parkinson disease (PD) patients, deep brain stimulation (DBS) of the subthalamic nucleus (STN) may contribute [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/16/the-subthalamic-nucleus-and-impulsivity/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Endocrine consequences of opioid therapy.</title>
		<link>http://www.definitivemind.com/2010/01/14/endocrine-consequences-of-opioid-therapy/</link>
		<comments>http://www.definitivemind.com/2010/01/14/endocrine-consequences-of-opioid-therapy/#comments</comments>
		<pubDate>Thu, 14 Jan 2010 21:07:39 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Hormones]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[adrenal]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[endocrine]]></category>
		<category><![CDATA[opioid]]></category>
		<category><![CDATA[testosterone]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=437</guid>
		<description><![CDATA[Endocrine consequences of opioid therapy. Psychoneuroendocrinology. 2009 Dec;34 Suppl 1:S162-8 Authors: Aloisi AM, Aurilio C, Bachiocco V, Biasi G, Fiorenzani P, Pace MC, Paci V, Pari G, Passavanti G, Ravaioli L, Sindaco G, Vellucci R, Ceccarelli I Gonadal hormones are known to be affected by morphine and other opioids. In this paper, we summarize data [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/14/endocrine-consequences-of-opioid-therapy/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Major depressive disorder and high cortisol levels: Textbook results that I hardly see in real life.</title>
		<link>http://www.definitivemind.com/2010/01/14/major-depressive-disorder-and-high-cortisol-levels-textbook-results-that-i-hardly-see-in-real-life/</link>
		<comments>http://www.definitivemind.com/2010/01/14/major-depressive-disorder-and-high-cortisol-levels-textbook-results-that-i-hardly-see-in-real-life/#comments</comments>
		<pubDate>Thu, 14 Jan 2010 08:10:33 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[TOPICS]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[major depression]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=433</guid>
		<description><![CDATA[Major depressive disorder and hypothalamic-pituitary-adrenal axis activity: results from a large cohort study.: &#8220; Related Articles Arch Gen Psychiatry. 2009 Jun;66(6):617-26 Authors: Vreeburg SA, Hoogendijk WJ, van Pelt J, Derijk RH, Verhagen JC, van Dyck R, Smit JH, Zitman FG, Penninx BW CONTEXT: There is a central belief that depression is associated with hyperactivity of [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/14/major-depressive-disorder-and-high-cortisol-levels-textbook-results-that-i-hardly-see-in-real-life/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>N-acetylcysteine, a glutamate modulator, in the treatment of trichotillomania</title>
		<link>http://www.definitivemind.com/2010/01/13/n-acetylcysteine-a-glutamate-modulator-in-the-treatment-of-trichotillomania/</link>
		<comments>http://www.definitivemind.com/2010/01/13/n-acetylcysteine-a-glutamate-modulator-in-the-treatment-of-trichotillomania/#comments</comments>
		<pubDate>Thu, 14 Jan 2010 06:56:22 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[glutamate]]></category>
		<category><![CDATA[N-acetylcysteine]]></category>
		<category><![CDATA[NAC]]></category>
		<category><![CDATA[trichotillomania]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=431</guid>
		<description><![CDATA[N-acetylcysteine, a glutamate modulator, in the treatment of trichotillomania: a double-blind, placebo-controlled study. Related Articles Arch Gen Psychiatry. 2009 Jul;66(7):756-63 Authors: Grant JE, Odlaug BL, Kim SW CONTEXT: Trichotillomania is characterized by repetitive hair pulling that causes noticeable hair loss. Data on the pharmacologic treatment of trichotillomania are limited to conflicting studies of serotonergic medications. [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/13/n-acetylcysteine-a-glutamate-modulator-in-the-treatment-of-trichotillomania/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Muscarinic Agonists for the Treatment of Cognition in Schizophrenia</title>
		<link>http://www.definitivemind.com/2010/01/13/muscarinic-agonists-for-the-treatment-of-cognition-in-schizophrenia/</link>
		<comments>http://www.definitivemind.com/2010/01/13/muscarinic-agonists-for-the-treatment-of-cognition-in-schizophrenia/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 09:28:51 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[acetylcholine]]></category>
		<category><![CDATA[cognition]]></category>
		<category><![CDATA[muscarinic]]></category>
		<category><![CDATA[schizophrenia]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=423</guid>
		<description><![CDATA[Muscarinic Agonists for the Treatment of Cognition in Schizophrenia Angela K. Sellin, MD, Mujeeb Shad, MD, and Carol Tamminga, MD CNS Spectr. 2008;13(11):985-996 It is widely accepted that cholinergic activity at muscarinic receptors is required to maintain cognitive functions, including learning and memory. Memory domains are especially impaired in schizophrenia, which may explain difficulties in [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/13/muscarinic-agonists-for-the-treatment-of-cognition-in-schizophrenia/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Normative variation in self-reported sleep quality and sleep debt is associated with stimulated pro-inflammatory cytokine production.</title>
		<link>http://www.definitivemind.com/2010/01/13/normative-variation-in-self-reported-sleep-quality-and-sleep-debt-is-associated-with-stimulated-pro-inflammatory-cytokine-production/</link>
		<comments>http://www.definitivemind.com/2010/01/13/normative-variation-in-self-reported-sleep-quality-and-sleep-debt-is-associated-with-stimulated-pro-inflammatory-cytokine-production/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 09:12:37 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cytokine]]></category>
		<category><![CDATA[proinflammatory]]></category>
		<category><![CDATA[sleep]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=420</guid>
		<description><![CDATA[Normative variation in self-reported sleep quality and sleep debt is associated with stimulated pro-inflammatory cytokine production. Related Articles Biol Psychol. 2009 Sep;82(1):12-7 Authors: Prather AA, Marsland AL, Hall M, Neumann SA, Muldoon MF, Manuck SB Activation of innate inflammatory pathways, marked by increased production of pro-inflammatory cytokines, has been proposed as a potential mechanism linking [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/13/normative-variation-in-self-reported-sleep-quality-and-sleep-debt-is-associated-with-stimulated-pro-inflammatory-cytokine-production/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Salivary gonadal and adrenal hormone differences in boys and girls with and without disruptive behavior disorders: Contextual variants.</title>
		<link>http://www.definitivemind.com/2010/01/13/salivary-gonadal-and-adrenal-hormone-differences-in-boys-and-girls-with-and-without-disruptive-behavior-disorders-contextual-variants/</link>
		<comments>http://www.definitivemind.com/2010/01/13/salivary-gonadal-and-adrenal-hormone-differences-in-boys-and-girls-with-and-without-disruptive-behavior-disorders-contextual-variants/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 09:10:31 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[adrenal]]></category>
		<category><![CDATA[disruptive behavior]]></category>
		<category><![CDATA[gonadal]]></category>
		<category><![CDATA[hormone]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=418</guid>
		<description><![CDATA[Salivary gonadal and adrenal hormone differences in boys and girls with and without disruptive behavior disorders: Contextual variants. Related Articles Biol Psychol. 2009 Apr;81(1):31-9 Authors: Dorn LD, Kolko DJ, Susman EJ, Huang B, Stein H, Music E, Bukstein OG Hormone differences by psychopathology group and gender may have implications for understanding disruptive behavior disorders (DBDs) [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/13/salivary-gonadal-and-adrenal-hormone-differences-in-boys-and-girls-with-and-without-disruptive-behavior-disorders-contextual-variants/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Transient responses of inflammatory cytokines in acute stress.</title>
		<link>http://www.definitivemind.com/2010/01/13/transient-responses-of-inflammatory-cytokines-in-acute-stress/</link>
		<comments>http://www.definitivemind.com/2010/01/13/transient-responses-of-inflammatory-cytokines-in-acute-stress/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 09:05:24 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cytokine]]></category>
		<category><![CDATA[proinflammatory]]></category>
		<category><![CDATA[stress]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=416</guid>
		<description><![CDATA[Transient responses of inflammatory cytokines in acute stress.: Related Articles Biol Psychol. 2009 Sep;82(1):25-32 Authors: Yamakawa K, Matsunaga M, Isowa T, Kimura K, Kasugai K, Yoneda M, Kaneko H, Ohira H It has been demonstrated that concentrations of pro-inflammatory cytokines such as interleukin-1beta (IL-1beta) and interleukin-6 (IL-6) are elevated by acute stress. Although several studies [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/13/transient-responses-of-inflammatory-cytokines-in-acute-stress/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Day-to-day variation in saliva cortisol&#8211;relation with sleep, stress and self-rated health.</title>
		<link>http://www.definitivemind.com/2010/01/13/day-to-day-variation-in-saliva-cortisol-relation-with-sleep-stress-and-self-rated-health/</link>
		<comments>http://www.definitivemind.com/2010/01/13/day-to-day-variation-in-saliva-cortisol-relation-with-sleep-stress-and-self-rated-health/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 09:01:26 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cortisol]]></category>
		<category><![CDATA[health]]></category>
		<category><![CDATA[sleep]]></category>
		<category><![CDATA[stress]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=414</guid>
		<description><![CDATA[Day-to-day variation in saliva cortisol&#8211;relation with sleep, stress and self-rated health. Related Articles Biol Psychol. 2009 Oct;82(2):149-55 Authors: Dahlgren A, Kecklund G, Theorell T, Akerstedt T The objective was to examine the day-to-day variation in cortisol among healthy individuals and its relation to the time of saliva sampling, work, stress and fatigue. During 4 consecutive [...]]]></description>
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		<title>Sleep physiology in recovery from burnout.</title>
		<link>http://www.definitivemind.com/2010/01/13/sleep-physiology-in-recovery-from-burnout/</link>
		<comments>http://www.definitivemind.com/2010/01/13/sleep-physiology-in-recovery-from-burnout/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 08:59:22 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[burnout]]></category>
		<category><![CDATA[sleep]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=412</guid>
		<description><![CDATA[Sleep physiology in recovery from burnout. Related Articles Biol Psychol. 2009 Dec;82(3):267-73 Authors: Ekstedt M, Söderström M, Akerstedt T This study aimed to investigate the role of sleep physiology in recovery from burnout, in particular the relation between sleep and changes in fatigue and whether those changes would be related to return to work. 23 [...]]]></description>
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		<title>Functional Disconnection of Frontal Cortex and Visual Cortex in Attention-Deficit/Hyperactivity Disorder.</title>
		<link>http://www.definitivemind.com/2010/01/13/functional-disconnection-of-frontal-cortex-and-visual-cortex-in-attention-deficithyperactivity-disorder/</link>
		<comments>http://www.definitivemind.com/2010/01/13/functional-disconnection-of-frontal-cortex-and-visual-cortex-in-attention-deficithyperactivity-disorder/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 08:53:38 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[ADHD]]></category>
		<category><![CDATA[attention]]></category>
		<category><![CDATA[deficit]]></category>
		<category><![CDATA[frontal cortex]]></category>
		<category><![CDATA[hyperactivity]]></category>
		<category><![CDATA[visual cortex]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=410</guid>
		<description><![CDATA[Functional Disconnection of Frontal Cortex and Visual Cortex in Attention-Deficit/Hyperactivity Disorder. Biol Psychiatry. 2010 Jan 6; Authors: Mazaheri A, Coffey-Corina S, Mangun GR, Bekker EM, Berry AS, Corbett BA BACKGROUND: Current pathophysiologic models of attention-deficit/hyperactivity disorder (ADHD) suggest that impaired functional connectivity within brain attention networks may contribute to the disorder. In this electroencephalographic (EEG) [...]]]></description>
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		<title>A beta(3)-Adrenergic-Leptin-Melanocortin Circuit Regulates Behavioral and Metabolic Changes Induced by Chronic Stress.</title>
		<link>http://www.definitivemind.com/2010/01/13/a-beta3-adrenergic-leptin-melanocortin-circuit-regulates-behavioral-and-metabolic-changes-induced-by-chronic-stress/</link>
		<comments>http://www.definitivemind.com/2010/01/13/a-beta3-adrenergic-leptin-melanocortin-circuit-regulates-behavioral-and-metabolic-changes-induced-by-chronic-stress/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 08:50:41 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=408</guid>
		<description><![CDATA[A beta(3)-Adrenergic-Leptin-Melanocortin Circuit Regulates Behavioral and Metabolic Changes Induced by Chronic Stress. Biol Psychiatry. 2010 Jan 8; Authors: Chuang JC, Krishnan V, Yu HG, Mason B, Cui H, Wilkinson MB, Zigman JM, Elmquist JK, Nestler EJ, Lutter M BACKGROUND: Obesity has been associated with an increased risk of developing several psychiatric illnesses, including major depression [...]]]></description>
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		<title>Auditory Hallucinations in Schizophrenia Are Associated with Reduced Functional Connectivity of the Temporo-Parietal Area.</title>
		<link>http://www.definitivemind.com/2010/01/13/auditory-hallucinations-in-schizophrenia-are-associated-with-reduced-functional-connectivity-of-the-temporo-parietal-area/</link>
		<comments>http://www.definitivemind.com/2010/01/13/auditory-hallucinations-in-schizophrenia-are-associated-with-reduced-functional-connectivity-of-the-temporo-parietal-area/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 08:47:13 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=406</guid>
		<description><![CDATA[Auditory Hallucinations in Schizophrenia Are Associated with Reduced Functional Connectivity of the Temporo-Parietal Area. Biol Psychiatry. 2010 Jan 6; Authors: Vercammen A, Knegtering H, den Boer JA, Liemburg EJ, Aleman A BACKGROUND: Schizophrenia has been conceptualized as a disorder of integration of neural activity across distributed networks. However, the relationship between specific symptom dimensions and [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/13/auditory-hallucinations-in-schizophrenia-are-associated-with-reduced-functional-connectivity-of-the-temporo-parietal-area/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
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		<title>Noradrenaline reuptake inhibitors inhibit expression of chemokines IP-10 and RANTES and cell adhesion molecules VCAM-1 and ICAM-1 in the CNS following a systemic inflammatory challenge.</title>
		<link>http://www.definitivemind.com/2010/01/12/noradrenaline-reuptake-inhibitors-inhibit-expression-of-chemokines-ip-10-and-rantes-and-cell-adhesion-molecules-vcam-1-and-icam-1-in-the-cns-following-a-systemic-inflammatory-challenge/</link>
		<comments>http://www.definitivemind.com/2010/01/12/noradrenaline-reuptake-inhibitors-inhibit-expression-of-chemokines-ip-10-and-rantes-and-cell-adhesion-molecules-vcam-1-and-icam-1-in-the-cns-following-a-systemic-inflammatory-challenge/#comments</comments>
		<pubDate>Wed, 13 Jan 2010 07:47:02 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=404</guid>
		<description><![CDATA[Noradrenaline reuptake inhibitors inhibit expression of chemokines IP-10 and RANTES and cell adhesion molecules VCAM-1 and ICAM-1 in the CNS following a systemic inflammatory challenge. Noradrenaline reuptake inhibitors inhibit expression of chemokines IP-10 and RANTES and cell adhesion molecules VCAM-1 and ICAM-1 in the CNS following a systemic inflammatory challenge. J Neuroimmunol. 2010 Jan 8; [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/12/noradrenaline-reuptake-inhibitors-inhibit-expression-of-chemokines-ip-10-and-rantes-and-cell-adhesion-molecules-vcam-1-and-icam-1-in-the-cns-following-a-systemic-inflammatory-challenge/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
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		<title>Decreased Frontal Serotonin2A Receptor Binding in Antipsychotic-Naive Patients With First-Episode Schizophrenia</title>
		<link>http://www.definitivemind.com/2010/01/11/decreased-frontal-serotonin2a-receptor-binding-in-antipsychotic-naive-patients-with-first-episode-schizophrenia/</link>
		<comments>http://www.definitivemind.com/2010/01/11/decreased-frontal-serotonin2a-receptor-binding-in-antipsychotic-naive-patients-with-first-episode-schizophrenia/#comments</comments>
		<pubDate>Tue, 12 Jan 2010 07:00:12 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[TOPICS]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=402</guid>
		<description><![CDATA[Decreased Frontal Serotonin2A Receptor Binding in Antipsychotic-Naive Patients With First-Episode Schizophrenia [Original Article] Arch Gen Psychiatry. 2010;67(1):9-16. Hans Rasmussen, PhD; David Erritzoe, MD, PhD; Rune Andersen, MSc; Bjorn H. Ebdrup, MD; Bodil Aggernaes, MD, PhD; Bob Oranje, PhD; Jan Kalbitzer, MD; Jacob Madsen, PhD; Lars H. Pinborg, MD, DMSc; William Baaré, PhD; Claus Svarer, PhD; [...]]]></description>
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		<title>Weight loss diet elevateds Interleukin-10 levels</title>
		<link>http://www.definitivemind.com/2010/01/11/weight-loss-diet-elevateds-interleukin-10-levels/</link>
		<comments>http://www.definitivemind.com/2010/01/11/weight-loss-diet-elevateds-interleukin-10-levels/#comments</comments>
		<pubDate>Mon, 11 Jan 2010 20:21:20 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cytokines]]></category>
		<category><![CDATA[interleukin-10]]></category>
		<category><![CDATA[obesity]]></category>
		<category><![CDATA[weight]]></category>

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		<description><![CDATA[Elevated blood interleukin-10 levels and undiminished systemic interleukin-10 production rate prevail throughout acute protein-energy malnutrition in the weanling mouse. Related Articles Elevated blood interleukin-10 levels and undiminished systemic interleukin-10 production rate prevail throughout acute protein-energy malnutrition in the weanling mouse. Cytokine. 2009 Aug;47(2):126-31 Authors: Monk JM, Woodward B The objectives were to determine if blood [...]]]></description>
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		<title>Exercise increases Interleukin-6/interleukin-6 receptor complex</title>
		<link>http://www.definitivemind.com/2010/01/11/exercise-increases-interleukin-6interleukin-6-receptor-complex/</link>
		<comments>http://www.definitivemind.com/2010/01/11/exercise-increases-interleukin-6interleukin-6-receptor-complex/#comments</comments>
		<pubDate>Mon, 11 Jan 2010 20:16:09 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cytokines]]></category>
		<category><![CDATA[exercise]]></category>
		<category><![CDATA[interleukin-6]]></category>

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		<description><![CDATA[The response of circulating levels of the interleukin-6/interleukin-6 receptor complex to exercise in young men. Related Articles The response of circulating levels of the interleukin-6/interleukin-6 receptor complex to exercise in young men. Cytokine. 2009 Aug;47(2):98-102 Authors: Gray SR, Clifford M, Lancaster R, Leggate M, Davies M, Nimmo MA The formation of an interleukin-6/IL-6 receptor (IL-6/IL-6R) [...]]]></description>
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		<slash:comments>0</slash:comments>
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		<title>Anxiety and outcome in bipolar disorder.</title>
		<link>http://www.definitivemind.com/2010/01/11/anxiety-and-outcome-in-bipolar-disorder/</link>
		<comments>http://www.definitivemind.com/2010/01/11/anxiety-and-outcome-in-bipolar-disorder/#comments</comments>
		<pubDate>Mon, 11 Jan 2010 19:54:55 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=396</guid>
		<description><![CDATA[Anxiety and outcome in bipolar disorder. Related Articles Anxiety and outcome in bipolar disorder. Am J Psychiatry. 2009 Nov;166(11):1238-43 Authors: Coryell W, Solomon DA, Fiedorowicz JG, Endicott J, Schettler PJ, Judd LL OBJECTIVE: Important differences exist between bipolar disorder with and without comorbid anxiety, but little is known about the long-term prognostic significance of coexisting [...]]]></description>
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		<title>Effect of acute antidepressant administration on negative affective bias in depressed patients.</title>
		<link>http://www.definitivemind.com/2010/01/11/effect-of-acute-antidepressant-administration-on-negative-affective-bias-in-depressed-patients/</link>
		<comments>http://www.definitivemind.com/2010/01/11/effect-of-acute-antidepressant-administration-on-negative-affective-bias-in-depressed-patients/#comments</comments>
		<pubDate>Mon, 11 Jan 2010 19:48:16 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Psychiatry]]></category>

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		<description><![CDATA[Effect of acute antidepressant administration on negative affective bias in depressed patients. Effect of acute antidepressant administration on negative affective bias in depressed patients. Am J Psychiatry. 2009 Oct;166(10):1178-84 Authors: Harmer CJ, O&#8217;Sullivan U, Favaron E, Massey-Chase R, Ayres R, Reinecke A, Goodwin GM, Cowen PJ OBJECTIVE: Acute administration of an antidepressant increases positive affective [...]]]></description>
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		</item>
		<item>
		<title>To Sleep, Perchance to Reset Your Body Clock</title>
		<link>http://www.definitivemind.com/2010/01/11/to-sleep-perchance-to-reset-your-body-clock/</link>
		<comments>http://www.definitivemind.com/2010/01/11/to-sleep-perchance-to-reset-your-body-clock/#comments</comments>
		<pubDate>Mon, 11 Jan 2010 08:58:45 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Hormones]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>

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		<description><![CDATA[To Sleep, Perchance to Reset Your Body Clock January 9, 2010 Psychiatric Times. Vol. 27 No. 2 Alfred J. Lewy, MD, PhD Dr Lewy is professor of psychiatry, ophthalmology, and physiology/pharmacology, senior vice chairman of the department of psychiatry, and director of the sleep and mood disorders laboratory at the Oregon Health &#038; Science University, [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Testosterone Augmentation Therapy Reduces Major Depressive Disorder Severity</title>
		<link>http://www.definitivemind.com/2010/01/10/testosterone-augmentation-therapy-reduces-major-depressive-disorder-severity/</link>
		<comments>http://www.definitivemind.com/2010/01/10/testosterone-augmentation-therapy-reduces-major-depressive-disorder-severity/#comments</comments>
		<pubDate>Mon, 11 Jan 2010 05:16:45 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Complementary Medicine]]></category>
		<category><![CDATA[Hormones]]></category>
		<category><![CDATA[Medications]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=390</guid>
		<description><![CDATA[Low-dose Transdermal Testosterone Augmentation Therapy Improves Depression Severity in Women Karen K. Miller, MD, Roy H. Perlis, MD, MSc, George I. Papakostas, MD, David Mischoulon, MD, PhD, Dan V. Iosifescu, MD, MSc, Danielle J. Brick, BA, and Maurizio Fava, MD CNS Spectr. 2009;14(12):688-694 [download PDF] Background: Inadequate response to antidepressant monotherapy in women with major [...]]]></description>
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		<item>
		<title>Urate as a predictor of the rate of clinical decline in Parkinson disease.</title>
		<link>http://www.definitivemind.com/2010/01/10/urate-as-a-predictor-of-the-rate-of-clinical-decline-in-parkinson-disease/</link>
		<comments>http://www.definitivemind.com/2010/01/10/urate-as-a-predictor-of-the-rate-of-clinical-decline-in-parkinson-disease/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 23:22:04 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=387</guid>
		<description><![CDATA[Urate as a predictor of the rate of clinical decline in Parkinson disease. Urate as a predictor of the rate of clinical decline in Parkinson disease. Arch Neurol. 2009 Dec;66(12):1460-8 Authors: Ascherio A, LeWitt PA, Xu K, Eberly S, Watts A, Matson WR, Marras C, Kieburtz K, Rudolph A, Bogdanov MB, Schwid SR, Tennis M, [...]]]></description>
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		<item>
		<title>Reduced function of the serotonin transporter is associated with decreased expression of BDNF</title>
		<link>http://www.definitivemind.com/2010/01/10/reduced-function-of-the-serotonin-transporter-is-associated-with-decreased-expression-of-bdnf/</link>
		<comments>http://www.definitivemind.com/2010/01/10/reduced-function-of-the-serotonin-transporter-is-associated-with-decreased-expression-of-bdnf/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 23:07:25 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>

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		<description><![CDATA[Reduced function of the serotonin transporter is associated with decreased expression of BDNF in rodents as well as in humans. Related Articles Reduced function of the serotonin transporter is associated with decreased expression of BDNF in rodents as well as in humans. Neurobiol Dis. 2009 Dec 23; Molteni R, Cattaneo A, Calabrese F, Macchi F, [...]]]></description>
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		<title>Chronic psychosocial stress triggers cognitive impairment in Alzheimer’s disease</title>
		<link>http://www.definitivemind.com/2010/01/10/chronic-psychosocial-stress-triggers-cognitive-impairment-in-alzheimer%e2%80%99s-disease/</link>
		<comments>http://www.definitivemind.com/2010/01/10/chronic-psychosocial-stress-triggers-cognitive-impairment-in-alzheimer%e2%80%99s-disease/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 23:01:28 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychology]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>

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		<description><![CDATA[Chronic psychosocial stress triggers cognitive impairment in a novel at-risk model of Alzheimer’s disease Neurobiology of Disease, In Press, Accepted Manuscript, Available online 4 January 2010Trinh T., Tran , Marisa, Srivareerat , Karim A., Alkadhi Abstract Although it is generally accepted that Aβ contributes to the pathogenesis of Alzheimer&#8217;s disease (AD), other factors that impact [...]]]></description>
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		<title>Interleukin-1beta impairs memory by reducing acetylcholine release and nerve growth factor expression, reversed by Omega-3 Fatty Acid EPA treatment</title>
		<link>http://www.definitivemind.com/2010/01/10/interleukin-1beta-impairs-memory-by-reducing-acetylcholine-release-and-nerve-growth-factor-expression-reversed-by-omega-3-fatty-acid-epa-treatment/</link>
		<comments>http://www.definitivemind.com/2010/01/10/interleukin-1beta-impairs-memory-by-reducing-acetylcholine-release-and-nerve-growth-factor-expression-reversed-by-omega-3-fatty-acid-epa-treatment/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 21:45:31 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[acetylcholine]]></category>
		<category><![CDATA[interleukin-1beta]]></category>
		<category><![CDATA[memory]]></category>
		<category><![CDATA[nerve growth factor]]></category>
		<category><![CDATA[omega-3]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=381</guid>
		<description><![CDATA[Reductions of acetylcholine release and nerve growth factor expression are correlated with memory impairment induced by interleukin-1beta administrations: effects of omega-3 fatty acid EPA treatment. Related Articles Reductions of acetylcholine release and nerve growth factor expression are correlated with memory impairment induced by interleukin-1beta administrations: effects of omega-3 fatty acid EPA treatment. J Neurochem. 2009 [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/10/interleukin-1beta-impairs-memory-by-reducing-acetylcholine-release-and-nerve-growth-factor-expression-reversed-by-omega-3-fatty-acid-epa-treatment/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
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		<title>Inhibiting p53 pathways in microglia attenuates microglial-evoked neurotoxicity following exposure to Alzheimer peptides.</title>
		<link>http://www.definitivemind.com/2010/01/10/inhibiting-p53-pathways-in-microglia-attenuates-microglial-evoked-neurotoxicity-following-exposure-to-alzheimer-peptides/</link>
		<comments>http://www.definitivemind.com/2010/01/10/inhibiting-p53-pathways-in-microglia-attenuates-microglial-evoked-neurotoxicity-following-exposure-to-alzheimer-peptides/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 21:12:56 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[TOPICS]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=379</guid>
		<description><![CDATA[Inhibiting p53 pathways in microglia attenuates microglial-evoked neurotoxicity following exposure to Alzheimer peptides.: &#8220; Related Articles Inhibiting p53 pathways in microglia attenuates microglial-evoked neurotoxicity following exposure to Alzheimer peptides. J Neurochem. 2009 Nov 6; Authors: Davenport CM, Sevastou IG, Hooper C, Pocock JM Microglial activation can lead to microglial apoptosis, which may serve to remove [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/10/inhibiting-p53-pathways-in-microglia-attenuates-microglial-evoked-neurotoxicity-following-exposure-to-alzheimer-peptides/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
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		<title>Is Heart Failure a Mental Illness? &#8211; MAO Inhibitors offers promise in treating heart failure</title>
		<link>http://www.definitivemind.com/2010/01/10/is-heart-failure-a-mental-illness-mao-inhibitors-offers-promise-in-treating-heart-failure/</link>
		<comments>http://www.definitivemind.com/2010/01/10/is-heart-failure-a-mental-illness-mao-inhibitors-offers-promise-in-treating-heart-failure/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 19:26:16 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Mind-Body Medicine]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[TOPICS]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=375</guid>
		<description><![CDATA[Is Heart Failure a mental illness? At the very least, it is a psychosomatic illness as this press release indicates. &#8212;&#8212;&#8212;- Public release date: 6-Jan-2010 Contact: David March dmarch1@jhmi.edu 410-955-1534 Johns Hopkins Medical Institutions Old antidepressant offers promise in treating heart failure Monoamine oxidase-A inhibitor drug blocks buildup of toxic free radicals in animal hearts [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/10/is-heart-failure-a-mental-illness-mao-inhibitors-offers-promise-in-treating-heart-failure/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
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		<title>Chronic Microglial Activation After an Inflammatory Event</title>
		<link>http://www.definitivemind.com/2010/01/10/chronic-microglial-activation-after-an-inflammatory-event/</link>
		<comments>http://www.definitivemind.com/2010/01/10/chronic-microglial-activation-after-an-inflammatory-event/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 19:11:50 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>
		<category><![CDATA[cytokines]]></category>
		<category><![CDATA[glia]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[microglia]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=373</guid>
		<description><![CDATA[Systemic lipopolysaccharide (LPS)-induced microglial activation results in different temporal reduction of CD200 and CD200 receptor gene expression in the brain. Related Articles Systemic lipopolysaccharide (LPS)-induced microglial activation results in different temporal reduction of CD200 and CD200 receptor gene expression in the brain. J Neuroimmunol. 2009 Sep 29;214(1-2):78-82 Authors: Masocha W LPS activates microglia, which are [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/10/chronic-microglial-activation-after-an-inflammatory-event/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Smoking Cessation Pharmacotherapy Does Not Work Well</title>
		<link>http://www.definitivemind.com/2010/01/10/smoking-cessation-pharmacotherapy-does-not-work-well/</link>
		<comments>http://www.definitivemind.com/2010/01/10/smoking-cessation-pharmacotherapy-does-not-work-well/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 18:59:28 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[TOPICS]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=369</guid>
		<description><![CDATA[Comparative Effectiveness of 5 Smoking Cessation Pharmacotherapies in Primary Care Clinics [Original Investigation] Stevens S. Smith, PhD; Danielle E. McCarthy, PhD; Sandra J. Japuntich, PhD; Bruce Christiansen, PhD; Megan E. Piper, PhD; Douglas E. Jorenby, PhD; David L. Fraser, MS; Michael C. Fiore, MD, MPH; Timothy B. Baker, PhD; Thomas C. Jackson, MD Arch Intern [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/10/smoking-cessation-pharmacotherapy-does-not-work-well/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
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		<title>Children who will grow up to develop adult schizophrenia struggle with verbal reasoning and lag in working memory, attention, and processing speed: A 30-Year Study.</title>
		<link>http://www.definitivemind.com/2010/01/10/children-who-will-grow-up-to-develop-adult-schizophrenia-struggle-with-verbal-reasoning-and-lag-in-working-memory-attention-and-processing-speed-a-30-year-study/</link>
		<comments>http://www.definitivemind.com/2010/01/10/children-who-will-grow-up-to-develop-adult-schizophrenia-struggle-with-verbal-reasoning-and-lag-in-working-memory-attention-and-processing-speed-a-30-year-study/#comments</comments>
		<pubDate>Sun, 10 Jan 2010 18:29:10 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Mental Health]]></category>
		<category><![CDATA[Neurobiology]]></category>
		<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=357</guid>
		<description><![CDATA[Static and Dynamic Cognitive Deficits in Childhood Preceding Adult Schizophrenia: A 30-Year Study. Am J Psychiatry. 2010 Jan 4; Authors: Reichenberg A, Caspi A, Harrington H, Houts R, Keefe RS, Murray RM, Poulton R, Moffitt TE OBJECTIVE: Premorbid cognitive deficits in schizophrenia are well documented and have been interpreted as supporting a neurodevelopmental etiological model. [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Abuse in childhood linked to migraine and other pain disorders</title>
		<link>http://www.definitivemind.com/2010/01/06/abuse-in-childhood-linked-to-migraine-and-other-pain-disorders/</link>
		<comments>http://www.definitivemind.com/2010/01/06/abuse-in-childhood-linked-to-migraine-and-other-pain-disorders/#comments</comments>
		<pubDate>Wed, 06 Jan 2010 08:09:29 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Mind-Body Medicine]]></category>
		<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychology]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=355</guid>
		<description><![CDATA[(Via EurekAlert! &#8211; Breaking News.) Abuse in childhood linked to migraine and other pain disorders: &#8220;Researchers from the American Headache Society&#8217;s Women&#8217;s Issues Section Research Consortium found that incidence of childhood maltreatment, especially emotional abuse and neglect, are prevalent in migraine patients. Full findings of the study appear in the January issue of Headache: The [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Leptin via Leptin-Linked Gene Reverses Diabetes</title>
		<link>http://www.definitivemind.com/2010/01/05/leptin-via-leptin-linked-gene-reverses-diabetes/</link>
		<comments>http://www.definitivemind.com/2010/01/05/leptin-via-leptin-linked-gene-reverses-diabetes/#comments</comments>
		<pubDate>Wed, 06 Jan 2010 02:55:07 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[TOPICS]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=351</guid>
		<description><![CDATA[Leptin-Linked Gene Reverses Diabetes in Mice (CME/CE): &#8220;The hunger hormone leptin can reverse diabetes (MedPage Today) &#8212; at least in mice &#8212; without causing weight loss, researchers have discovered.&#8221; Antidiabetic Effects of IGFBP2, a Leptin-Regulated Gene Cell Metabolism, Volume 11, Issue 1, 11-22, 6 January 2010 Kristina Hedbacker1, Kıvanç Birsoy1, Robert W. Wysocki1, 2, Esra [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/05/leptin-via-leptin-linked-gene-reverses-diabetes/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Metabolic Tests Ignored in Medicaid Patients on Antipsychotics</title>
		<link>http://www.definitivemind.com/2010/01/05/metabolic-tests-ignored-in-medicaid-patients-on-antipsychotics/</link>
		<comments>http://www.definitivemind.com/2010/01/05/metabolic-tests-ignored-in-medicaid-patients-on-antipsychotics/#comments</comments>
		<pubDate>Tue, 05 Jan 2010 21:08:53 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Medications]]></category>
		<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=345</guid>
		<description><![CDATA[Metabolic Tests Ignored in Medicaid Patients on Antipsychotics (CME/CE): &#8220;Antipsychotic-induced metabolic disturbances receive little attention among Medicaid patients treated with second-generation antipsychotics, according to an analysis of more than 100,000 cases.&#8221; Antipsychotic-induced metabolic disturbances receive little attention among Medicaid patients treated with second-generation drugs, according to an analysis of more than 100,000 cases. Slightly more [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Vitamin D insufficiency and health outcomes over 5 y in older women.</title>
		<link>http://www.definitivemind.com/2010/01/05/vitamin-d-insufficiency-and-health-outcomes-over-5-y-in-older-women/</link>
		<comments>http://www.definitivemind.com/2010/01/05/vitamin-d-insufficiency-and-health-outcomes-over-5-y-in-older-women/#comments</comments>
		<pubDate>Tue, 05 Jan 2010 14:42:25 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Nutrition]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[TOPICS]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=343</guid>
		<description><![CDATA[Vitamin D insufficiency and health outcomes over 5 y in older women. Related Articles Vitamin D insufficiency and health outcomes over 5 y in older women. Am J Clin Nutr. 2010 Jan;91(1):82-9 Authors: Bolland MJ, Bacon CJ, Horne AM, Mason BH, Ames RW, Wang TK, Grey AB, Gamble GD, Reid IR BACKGROUND: Vitamin D insufficiency [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Prescribing Buprenorphine May Trigger Random Federal Audit [Government News]</title>
		<link>http://www.definitivemind.com/2010/01/05/prescribing-buprenorphine-may-trigger-random-federal-audit-government-news/</link>
		<comments>http://www.definitivemind.com/2010/01/05/prescribing-buprenorphine-may-trigger-random-federal-audit-government-news/#comments</comments>
		<pubDate>Tue, 05 Jan 2010 14:26:15 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=341</guid>
		<description><![CDATA[Prescribing Buprenorphine May Trigger Random Federal Audit [Government News] The Drug Enforcement Administration (DEA) has recently sent letters to psychiatrists and other physicians who are registered with the DEA stating that it will be conducting on-site, unannounced inspections under the authority of the Controlled Substances Act (CSA). The inspections are intended to ensure compliance with [...]]]></description>
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		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Ovarian hormones (i.e. estrogen and progesterone)  on genes promoting dendritic spines in laser-captured serotonin neurons</title>
		<link>http://www.definitivemind.com/2010/01/05/ovarian-hormones-ie-estrogen-and-progesterone-on-genes-promoting-dendritic-spines-in-laser-captured-serotonin-neurons/</link>
		<comments>http://www.definitivemind.com/2010/01/05/ovarian-hormones-ie-estrogen-and-progesterone-on-genes-promoting-dendritic-spines-in-laser-captured-serotonin-neurons/#comments</comments>
		<pubDate>Tue, 05 Jan 2010 14:21:59 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=339</guid>
		<description><![CDATA[Effect of ovarian hormones on genes promoting dendritic spines in laser-captured serotonin neurons from macaques. Related Articles Effect of ovarian hormones on genes promoting dendritic spines in laser-captured serotonin neurons from macaques. Mol Psychiatry. 2009 Aug 18; Authors: Bethea CL, Reddy AP Dendritic spines are the elementary structural units of neuronal plasticity and the cascades [...]]]></description>
		<wfw:commentRss>http://www.definitivemind.com/2010/01/05/ovarian-hormones-ie-estrogen-and-progesterone-on-genes-promoting-dendritic-spines-in-laser-captured-serotonin-neurons/feed/</wfw:commentRss>
		<slash:comments>0</slash:comments>
		</item>
		<item>
		<title>Neurobiological trait abnormalities in bipolar disorder.</title>
		<link>http://www.definitivemind.com/2010/01/05/neurobiological-trait-abnormalities-in-bipolar-disorder/</link>
		<comments>http://www.definitivemind.com/2010/01/05/neurobiological-trait-abnormalities-in-bipolar-disorder/#comments</comments>
		<pubDate>Tue, 05 Jan 2010 14:18:23 +0000</pubDate>
		<dc:creator>Romeo Mariano MD</dc:creator>
				<category><![CDATA[Neuro-endocrinology]]></category>
		<category><![CDATA[Psychiatry]]></category>
		<category><![CDATA[Psychoneuro-immunology]]></category>

		<guid isPermaLink="false">http://www.definitivemind.com/?p=337</guid>
		<description><![CDATA[Neurobiological trait abnormalities in bipolar disorder. Related Articles Neurobiological trait abnormalities in bipolar disorder. Mol Psychiatry. 2009 Sep;14(9):833-46 Authors: Langan C, McDonald C Dissecting trait neurobiological abnormalities in bipolar disorder (BD) from those characterizing episodes of mood disturbance will help elucidate the aetiopathogenesis of the illness. This selective review highlights the immunological, neuroendocrinological, molecular biological [...]]]></description>
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		<slash:comments>0</slash:comments>
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